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1. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 7, 2011, No. 2 , , ,

Hemineglect, Hemianopsia and Signs of Gerstmann Syndrome in Brain Lobar Parenchymal Hemorrhage –
Hemineglect, Hemianopsia and Signs of Gerstmann Syndrome in Brain Lobar Parenchymal Hemorrhage –
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Hemineglect, Hemianopsia and Signs of Gerstmann Syndrome in Brain Lobar Parenchymal Hemorrhage –
Hemineglect, Hemianopsia and Signs of Gerstmann Syndrome in Brain Lobar Parenchymal Hemorrhage –
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brain hemorrhage, Gerstmann syndrome, hemianopsia, hemineglect
brain hemorrhage, Gerstmann syndrome, hemianopsia, hemineglect
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Neuropsychological assessment confirmed cardinal signs of Gerstmann syndrome and visual-spatial neglect.
The neurological status revealed right homonymous hemianopsia, anisoreflexia and right visual-spatial neglect. Finger agnosia; acalculia, left-right disorientation and constructive apraxia, alexia without agrafia were found. Hemianopsia was objectified by perimetry and visual evoked potentials. Neuropsychological assessment confirmed cardinal signs of Gerstmann syndrome and visual-spatial neglect.
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Higher cortical functions were impaired – cardinal signs of Gerstmann syndrome and right unilateral neglect were found.
Neurological examination revealed right homonymous hemianopsia, right pyramidal signs and right facial and truncal hemihypesthesia to touch and pain. Higher cortical functions were impaired – cardinal signs of Gerstmann syndrome and right unilateral neglect were found.
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Finger agnosia, acalculia, left-right disorientation, constructive apraxia and alexia without agraphia represent the cardinal signs of Gerstmann syndrome with right hemineglect manifestation.
Finger agnosia, acalculia, left-right disorientation, constructive apraxia and alexia without agraphia represent the cardinal signs of Gerstmann syndrome with right hemineglect manifestation.
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We observed the rare combination of hemineglect and hemianopsia, as well as Gerstmann syndrome due to brain hemorrhage in the left parietooccipital lobe.
The presented case is interesting because of its complex combination and difficult differentiation of simultaneously occurring visual and visual-spatial disturbances. We observed the rare combination of hemineglect and hemianopsia, as well as Gerstmann syndrome due to brain hemorrhage in the left parietooccipital lobe. Similar results are revealed by other authors [3, 5, 7, 10].
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It was found that this type of disorder could be the anatomic substrate of Gerstmann syndrome [8, 13, 16].
rietalis inferior of the left hemisphere are still discussed. When this region is damaged the patients have difficulties in forming letters due to limited or no access to the engrams. It was found that this type of disorder could be the anatomic substrate of Gerstmann syndrome [8, 13, 16].
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Gerstmann syndrome rarely occurs in pure form, which causes disputes for its localization importance.
In 1940 Josef Gerstmann united the symptoms finger agnosia, acalculia, agrafia and leftright disorientation in a single syndrome. He considered that the syndrome had a localization value and was a result from damage of the parietal lobe of dominant for language functions hemisphere – left gyrus angularis in particular. According to Gerstmann the syndrome was a result of the "unique fingerprint sense" impairment and the presence of the other symptoms derived from it [15, 16]. Gerstmann syndrome rarely occurs in pure form, which causes disputes for its localization importance. Its clinical expression is usually accompanied by additional symptoms: alexia, amnesic aphasia, constructive apraxia, right-sided central hemipalsy, right homonymous hemianopsia, forming the extended Gerstmann syndrome [2]. For differentiation of cardinal features of Gerstmann syndrome a target neuropsychological testing with gnosis, praxis and language functions monitoring is applied.
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Its clinical expression is usually accompanied by additional symptoms: alexia, amnesic aphasia, constructive apraxia, right-sided central hemipalsy, right homonymous hemianopsia, forming the extended Gerstmann syndrome [2].
In 1940 Josef Gerstmann united the symptoms finger agnosia, acalculia, agrafia and leftright disorientation in a single syndrome. He considered that the syndrome had a localization value and was a result from damage of the parietal lobe of dominant for language functions hemisphere – left gyrus angularis in particular. According to Gerstmann the syndrome was a result of the "unique fingerprint sense" impairment and the presence of the other symptoms derived from it [15, 16]. Gerstmann syndrome rarely occurs in pure form, which causes disputes for its localization importance. Its clinical expression is usually accompanied by additional symptoms: alexia, amnesic aphasia, constructive apraxia, right-sided central hemipalsy, right homonymous hemianopsia, forming the extended Gerstmann syndrome [2]. For differentiation of cardinal features of Gerstmann syndrome a target neuropsychological testing with gnosis, praxis and language functions monitoring is applied.
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For differentiation of cardinal features of Gerstmann syndrome a target neuropsychological testing with gnosis, praxis and language functions monitoring is applied.
In 1940 Josef Gerstmann united the symptoms finger agnosia, acalculia, agrafia and leftright disorientation in a single syndrome. He considered that the syndrome had a localization value and was a result from damage of the parietal lobe of dominant for language functions hemisphere – left gyrus angularis in particular. According to Gerstmann the syndrome was a result of the "unique fingerprint sense" impairment and the presence of the other symptoms derived from it [15, 16]. Gerstmann syndrome rarely occurs in pure form, which causes disputes for its localization importance. Its clinical expression is usually accompanied by additional symptoms: alexia, amnesic aphasia, constructive apraxia, right-sided central hemipalsy, right homonymous hemianopsia, forming the extended Gerstmann syndrome [2]. For differentiation of cardinal features of Gerstmann syndrome a target neuropsychological testing with gnosis, praxis and language functions monitoring is applied.
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The study shows that the diagnosis of enlarged Gerstmann syndrome with hemianopsia and hemineglect requires a multidisciplinary approach and is supported by the combined use of clinical, neuropsychological, neurophysiologic and ophthalmological examinations.
The study shows that the diagnosis of enlarged Gerstmann syndrome with hemianopsia and hemineglect requires a multidisciplinary approach and is supported by the combined use of clinical, neuropsychological, neurophysiologic and ophthalmological examinations. The prospective monitoring is important. Application of neuroimaging methods contributes to objectify the location, severity and type of damage in the brain parenchyma and its evolution.
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J. A pure case of Gerstmann syndrome with a subangular
J. A pure case of Gerstmann syndrome with a subangular
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