Search for 'anticoagulant therapy'

Search in texts for 'anticoagulant therapy' - Neurosonology.net'

Download page (1)

12 results of 7 texts with exact phrase : 'anticoagulant therapy'.

1. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 2, 2006, No. 2 , , ,

CVO syndrome management aims anticoagulant therapy and control of the intracranial pressure.
Cerebral venous occlusion (CVO) is unusual clinical event with various pathological manifestations. Appearance of clinical signs and extent of affected anatomical structures are related with death frequency, appr. 60%. Cascade development of the syndrome appears with focal neurological deficit, increased intracranial pressure, gradually alteration of the conscience and seizures. Computed tomography (CT) and cerebrospinal fluid (CSF) investigations (sample) are main tools of diagnostics. CVO syndrome management aims anticoagulant therapy and control of the intracranial pressure.
read the entire text >>

CT angiography is of great value for diagnosis of CVO and estimation of the cerebral venous system as far as for evolution of effectiveness of the anticoagulant therapy.
Two patients who underwent craniocerebral gunshot injuries with direct damage of venous sinuses and venous sinuses thrombosis with good outcome are presented. CT angiography is of great value for diagnosis of CVO and estimation of the cerebral venous system as far as for evolution of effectiveness of the anticoagulant therapy.
read the entire text >>

Good final results of treatment, without any changes in sinus thrombosis is due to rapid compensation of venous blood flow by anastomoses, “intracranial decompression” by CSF and cerebral detritus leakage, surgical and therapeutical control of the brain edema, despite of lack of effect of anticoagulant therapy in one of the patients.
Good final results of treatment, without any changes in sinus thrombosis is due to rapid compensation of venous blood flow by anastomoses, “intracranial decompression” by CSF and cerebral detritus leakage, surgical and therapeutical control of the brain edema, despite of lack of effect of anticoagulant therapy in one of the patients.
read the entire text >>

2. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 4, 2008, No. 1 , , ,

dignosis and anticoagulant therapy.
dignosis and anticoagulant therapy.
read the entire text >>

3. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 10, 2014, No. 2 , , ,

Starting anticoagulant therapy.
Starting anticoagulant therapy.
read the entire text >>

4. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 11, 2015, No. 1 , , ,

They are defined as partial or full hemophthalm and often associated with the wrong dosage of anticoagulant therapy.
Intraocular hemorrhages (hemophthalm) are polietiologic (diabetes, atherosclerosis, hypertension, antikoyagulantno treatment and others.). They are defined as partial or full hemophthalm and often associated with the wrong dosage of anticoagulant therapy. The changes are heterogeneous and predominantly located in the posterior segment of the vitreous and are age-depended of the bleeding in the subacute stage they are variable hyperechogenic artifacts and could be absorbed partially or in full (fig. 7).
read the entire text >>

5. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 11, 2015, No. 2 , , ,

Multidisciplinary team of a neurologist, cardiologist and transfusiologist discussed over the decision whether to use anticoagulant therapy.
Initial brain CT showed a small post-ischemic zone in the left parietal lobe. Doppler ultrasound of carotid arteries was normal. A repeated CT brain after 48 hours showed a large left middle cerebral artery infarction with focal areas of hemorrhagic transformation. Echocardiography revealed a septoapical aneurysm with akinetic walls, a mobile thrombus in the apical region and thrombi in the left ventricle. Brain MRI confirmed the CT scan finding. Multidisciplinary team of a neurologist, cardiologist and transfusiologist discussed over the decision whether to use anticoagulant therapy.
read the entire text >>

6. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 12, 2016, No. 2 , , ,

Although the use of anticoagulant therapy is controversial, many authors suggest dose-adjusted intravenous heparin applications if there are no contraindications, such as risk of intracranial or any other hemorrhage [15].
1n all cases empiric treatment with broad spectrum antibiotics is recommended, because infection is one of the most common causes of SOVT; later on antibiotics could be changed according to the antibiogram. Antibiotics should be given for 2 more weeks after clinical resolution, because pathogens can be located and sequestered within the thrombus [14]. Since in our case maxillary teeth extractions were the probable trigger factor for SOVT, we started treatment with dual broad spectrum antibiotics, which fortunately proved to be successful. The role of anticoagulants in SOVT cases is unclear. On the other hand, if not treated, SOVT can progress to cavernous sinus thrombosis. Although the use of anticoagulant therapy is controversial, many authors suggest dose-adjusted intravenous heparin applications if there are no contraindications, such as risk of intracranial or any other hemorrhage [15]. The EFNS guidelines for treatment of cerebral venous and sinus thrombosis in adults recommend body weightadjusted subcutaneous LMWH or dose-adjusted intravenous heparin use with an at least doubled activated partial thromboplastin time [4]. A metaanalysis comparing the efficacy of adjusted-dose unfractionated heparin and fixed-dose subcutaneous LMWH for extracerebral venous thromboembolism found superiority for LMWH and significantly less major bleeding complications [4, 16]. Guided by these recommendations we started the therapy with LMWH. Einhapul et al. suggest maintaining an elevated РTT in SOVT patients, but no more than
read the entire text >>

Well defined risk factors for ICH are age, alcohol consumption, hypertension, cerebral amyloid angiopathy, anticoagulant therapy, hemorrhagic transformation of ischemic stroke, vascular abnormalities, venous thrombosis, vasculitis, coagulopathy, and neoplasia.
Nontraumatic intracerebral hemorrhage (ICH) leads to a high rate of morbidity and mortality and constitutes a major public health problem worldwide, accounting for 10%– 15% of all strokes each year. Well defined risk factors for ICH are age, alcohol consumption, hypertension, cerebral amyloid angiopathy, anticoagulant therapy, hemorrhagic transformation of ischemic stroke, vascular abnormalities, venous thrombosis, vasculitis, coagulopathy, and neoplasia. Although CT is the first-line diagnostic approach, MR imaging with gradient echo sequences can detect hyperacute ICH with equal sensitivity and overall accuracy. Furthermore, MR imaging is more accurate for the detection of micro-hemorrhages.
read the entire text >>

Patients with ICH whose INR is elevated due to oral anticoagulant therapy should have their warfarin withheld, receive therapy to replace vitamin K–dependent factors and correct the INR, and receive intravenous vitamin K.
Patients with a severe coagulation factor deficiency or severe thrombocytopenia should receive appropriate factor replacement therapy or platelets, respectively. Patients with ICH whose INR is elevated due to oral anticoagulant therapy should have their warfarin withheld, receive therapy to replace vitamin K–dependent factors and correct the INR, and receive intravenous vitamin K.
read the entire text >>

7. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 14, 2018, No. 2 , , ,

For children with sinovenous thrombosis or arterial stroke due to dissection or cardiac embolism guidelines recommend anticoagulant therapy with warfarin or low molecular weight heparin for 3-6 months.
applicable to pediatric stroke due to maturational differences in coagulation and vascular systems as well as different stroke mechanisms. Current guidelines agree on the treatment of children with sickle-cell disease and stroke – initial and maintenance transfusion therapy to reduce the proportion of sickle-cell haemoglobin to less than 30%. For children with sinovenous thrombosis or arterial stroke due to dissection or cardiac embolism guidelines recommend anticoagulant therapy with warfarin or low molecular weight heparin for 3-6 months. Children with ischemic stroke of other etiology are empirically treated with antithrombotics including antiplatelet and anticoagulant drugs. Of major concern is also the risk of recurrent stroke, which affects up to 25% of children.
read the entire text >>

anticoagulant therapy, ischemia of the legs, left ventricle apical mobile thrombus
anticoagulant therapy, ischemia of the legs, left ventricle apical mobile thrombus
read the entire text >>

Back to Top