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176 results of 22 texts with exact phrase : 'autonomic'.

1. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, Vol. 1, 2005 , , ,

Methods for quantitative evaluation of the autonomic nervous system.
Hilz M, Dutsch M. Methods for quantitative evaluation of the autonomic nervous system.
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Orthostatic dysregulation in progressive autonomic failure: a transcranial Doppler sonographiy monitoring.
Titinova E, Karakaneva S, Velcheva I. Orthostatic dysregulation in progressive autonomic failure: a transcranial Doppler sonographiy monitoring.
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Prevalence of diabetic autonomic neuropathy measured by simple bedside tests.
Dyrberg T, Benn J, Christiansen J, Hilsted J, Nerup J. Prevalence of diabetic autonomic neuropathy measured by simple bedside tests.
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Diagnosing Diabetic Autonomic Neuropathy.
Vinik AI. Diagnosing Diabetic Autonomic Neuropathy.
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2. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 1, 2005, No. 2 , , ,

Autonomic control of heart rate during exercise studies by heart rate varriabiliti spectral analisis.
Yamamoto Y, Hughson R, Peterson J. Autonomic control of heart rate during exercise studies by heart rate varriabiliti spectral analisis.
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Effects of treatment with antioxidant alpha-lipoic acid cardiac autonomic neuropathy in NIDDM, patients: a 4month random.
Ziegler D. Effects of treatment with antioxidant alpha-lipoic acid cardiac autonomic neuropathy in NIDDM, patients: a 4month random. Controlled multicenter trial |DEKAN Study|.
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3. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 2, 2006, No. 1 , , ,

Relationship between phasic changes in human skin blood flow and autonomic tone.
Bernardi L, Rossi M, Fratino P, Finardi G, Mevio E, Orlandi C. Relationship between phasic changes in human skin blood flow and autonomic tone.
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4. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 2, 2006, No. 2 , , ,

Cardiovascular Autonomic Dysfunctions in
Cardiovascular Autonomic Dysfunctions in
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A new approach to the diagnosis of diabetic peripheral autonomic neuropathy.
Aso Y, Inukai T, Takemura Y. Evaluation of skin vasomotor reflexes in response to deep inspiration in diabetic patients by laser Doppler flowmetry. A new approach to the diagnosis of diabetic peripheral autonomic neuropathy.
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Electrophysiological evaluation of peripheral autonomic function in leprosy patients, leprosy contacts and controls.
Wilder-Smith A, Wilder-Smith E. Electrophysiological evaluation of peripheral autonomic function in leprosy patients, leprosy contacts and controls.
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autonomic dysfunctions, cardiovascular, diabetic polyneuropathy
autonomic dysfunctions, cardiovascular, diabetic polyneuropathy
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Cardiovascular Autonomic Dysfunctions in Diabetic Polyneuropathy
Cardiovascular Autonomic Dysfunctions in Diabetic Polyneuropathy
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To study the cardiovascular autonomic dysfunctions in diabetic polineuropathy.
To study the cardiovascular autonomic dysfunctions in diabetic polineuropathy.
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The cardiovascular autonomic dysfunctions were estimated as heart rate responses to different stimuli: deep breathing, the Valsalva manoeuvre and active standing.
The study included 22 patients (15 women, 7 men, mean age 55,52±13,75 years) with diabetic polyneuropathy and 22 healthy subjects. The cardiovascular autonomic dysfunctions were estimated as heart rate responses to different stimuli: deep breathing, the Valsalva manoeuvre and active standing. The R-R intervals were recorded by means of MP100 computerizes system with ECG module. Detection of R
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The data obtained correlate with the results from the power spectral analysis of the autonomic balance and increase its sensitivity.
The data obtained correlate with the results from the power spectral analysis of the autonomic balance and increase its sensitivity.
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Autonomic Dysfunctions in Diabetic Polyneuropathy
Autonomic Dysfunctions in Diabetic Polyneuropathy
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Autonomic Dysfunctions in Diabetic Polyneuropathy
Autonomic Dysfunctions in Diabetic Polyneuropathy
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Autonomic neuropathy in non insulin dependant (type II) diabetes mel� litus.
Bergstrom B, Lilja B, Osterlin S, Sundqvist, G. Autonomic neuropathy in non insulin dependant (type II) diabetes mel� litus. Possible influence of obesity.
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Cardiac autonomic neuropathy predicts cardio� vascular morbidity and mortality in type 1 diabetic patients with diabetic nephropathy.
Astrup AS, Tarnow L, Rossing P, Hansen BV, Hilsted J, Parving HH. Cardiac autonomic neuropathy predicts cardio� vascular morbidity and mortality in type 1 diabetic patients with diabetic nephropathy.
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Time and frequency domainestimatesofspontaneousbaroreflexsensitivity provide early detection of autonomic dysfunction in diabetes mellitus.
Frattola A, Parati G, Gamba P. et al. Time and frequency domainestimatesofspontaneousbaroreflexsensitivity provide early detection of autonomic dysfunction in diabetes mellitus.
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Spectral analysis of heart rate in diabetic autonomic neuropathy.
Freeman R. Saul J, Roberts M, Berger R, Broadbridge C and Cohen R. Spectral analysis of heart rate in diabetic autonomic neuropathy. A comparison with standart tests of autonomic function.
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A comparison with standart tests of autonomic function.
Freeman R. Saul J, Roberts M, Berger R, Broadbridge C and Cohen R. Spectral analysis of heart rate in diabetic autonomic neuropathy. A comparison with standart tests of autonomic function.
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The value of cardio� vascular autonomic function tests: 10 years experience in diabetes.
Ewing D, Martyn C, Young R, Clarke B. The value of cardio� vascular autonomic function tests: 10 years experience in diabetes.
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time of day in healthy controls and comparison with bedside tests of autonomic function in diabetic patients.
time of day in healthy controls and comparison with bedside tests of autonomic function in diabetic patients.
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Cardiovascular autonomic nervous system tests: Determination of norma� tive values and effect of confounding variables.
Gelber D, Pfeifer M, Dawson B, Schumer M. Cardiovascular autonomic nervous system tests: Determination of norma� tive values and effect of confounding variables.
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J Autonomic Nervous System
J Autonomic Nervous System
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Diabetic autonomic neuropathy.
Hilsted J, Low P. Diabetic autonomic neuropathy. In: Ph. A. Low (eds). Clinical autonomic disorders. Lippincott�Raven Publishers. Philadelphia � New York, 1997, 487�508.
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Clinical autonomic disorders.
Hilsted J, Low P. Diabetic autonomic neuropathy. In: Ph. A. Low (eds). Clinical autonomic disorders. Lippincott�Raven Publishers. Philadelphia � New York, 1997, 487�508.
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Autonomic neuropathy in a diabetic population.
Latini A, Martin L, Limiti G, Magarelli M, Polidori L, Tramutoli R, Papini E, Rinaldi R, Guglielmi R, Petrucci L, Panunzi C, Pagano, A. Autonomic neuropathy in a diabetic population. Validity of cardiovascular tests and correlations with the complications.
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Laboratory evaluation of autonomic function.
Low Ph. Laboratory evaluation of autonomic function. In: Ph.
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Clinical autonomic disorders.
A. Low (eds). Clinical autonomic disorders. Lippincott�Raven Publishers. Philadelphia � New York, 1997:179�208.
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The Association Between Cardiovascular Autonomic Neuropathy and Mortality in Individuals With Diabetes.
Maser R, Mitchell B, Vinik A, Freeman R. The Association Between Cardiovascular Autonomic Neuropathy and Mortality in Individuals With Diabetes. A meta�analysis.
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Autonomic neuroscience: basic and clinical
Autonomic neuroscience: basic and clinical
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P. Progression of diabetic autonomic neuropathy over a decade in insulin� dependant diabetics.
P. Progression of diabetic autonomic neuropathy over a decade in insulin� dependant diabetics.
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Diabetic Autonomic Neuropathy.
Vinik A, Maser R, Mitchell B, Freeman R. Diabetic Autonomic Neuropathy.
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Cardiovascular reflexes and autonomic neuropathy.
Ewing D. Cardiovascular reflexes and autonomic neuropathy.
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autonomic neuropathy and QT interval length: a follow-up study in diabetic patiens.
autonomic neuropathy and QT interval length: a follow-up study in diabetic patiens.
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Recognizing and treating diabetic autonomic neuropathy.
Vinik A, Erbas T. Recognizing and treating diabetic autonomic neuropathy.
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5. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 4, 2008, No. 1 , , ,

Clinical Autonomic Research
Clinical Autonomic Research
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6. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 5, 2009, No. 1 , , ,

Reactivity in Distal Autonomic Neuropathy
Reactivity in Distal Autonomic Neuropathy
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Clinical Autonomic Research
Clinical Autonomic Research
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and functional autonomic tests: heating, mild cooling, postural venoarteriolar in dependency and above heart level, reactive hyperemia test.
and functional autonomic tests: heating, mild cooling, postural venoarteriolar in dependency and above heart level, reactive hyperemia test. Skin vasomotor responses were assessed by analyzing perfusion values and constrictor and dilatator indices – delta perfusion (ΔLDF) and percent change (LDF%). In and between group comparisons were made by Mann-Whitney U rank test and Wilcoxon test for independent and dependent variables using SPSS software package.
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Vasomor reactivity in distal autonomic neuropathy
Vasomor reactivity in distal autonomic neuropathy
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Vasomor reactivity in distal autonomic neuropathy
Vasomor reactivity in distal autonomic neuropathy
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Vasomor reactivity in distal autonomic neuropathy
Vasomor reactivity in distal autonomic neuropathy
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Autonomic nervous function assessment using thermal reactivity of microcirculation.
Bonelli RM, Koltringer P. Autonomic nervous function assessment using thermal reactivity of microcirculation.
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Autonomic impairment in painful neuropathy.
Novak V, Freimer ML, Kissel JT, Sahenk Z, Periquet IM, Nash SM, Collins MP, Mendell JR. Autonomic impairment in painful neuropathy.
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Electrophysiological evaluation of peripheral autonomic function in leprosy patients, leprosy contacts and controls.
Wilder-Smith A, Wilder-Smith E. Electrophysiological evaluation of peripheral autonomic function in leprosy patients, leprosy contacts and controls.
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Greening J, Lynn B, Leary R Sensory and autonomic function in the hands of patients with non-specific arm pain (NSAP) and asymptomatic office workers.
Greening J, Lynn B, Leary R Sensory and autonomic function in the hands of patients with non-specific arm pain (NSAP) and asymptomatic office workers.
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Idiopathic autonomic neuropathy: Clinical, neurophysiologic, and follow-up studies on 27 patients.
Suarez GA, Fealey RD, Camilleri M, Low PA. Idiopathic autonomic neuropathy: Clinical, neurophysiologic, and follow-up studies on 27 patients.
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7. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 5, 2009, No. 1 , , ,

Combined immunomodulatory therapy in autoimmune autonomic ganglionopathy.
Gibbons CH, Vernino SA, Freeman R. Combined immunomodulatory therapy in autoimmune autonomic ganglionopathy.
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Subacute sensory neuronopathy is the most frequent presentation of paraneoplastic neuropathies, but sensory-motor neuropathy, autonomic neuropathy and mononeuritis multiplex with vasculitis have been also described.
Paraneoplastic neuropathies are rare diseases related to underlying neoplasma where immunological mechanisms are involved – immune response to onconeural antigens. Many anti-neuronal antibodies have been identified but most of them are not specific except for anti-Hu-antibodies. Subacute sensory neuronopathy is the most frequent presentation of paraneoplastic neuropathies, but sensory-motor neuropathy, autonomic neuropathy and mononeuritis multiplex with vasculitis have been also described. Sometimes the diagnosis is very difficult and includes different imaging studies, laboratory, electromyographic and histologic tests. Diagnostic criteria have been established and divide the patients into two groups: with definite and with probable diagnosis. The treatment of these diseases is complex and depends on the characteristics of primary neoplasm and neuropathy.
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Combined immunomodulatory therapy in autoimmune autonomic ganglionopathy.
Gibbons CH, Vernino SA, Freeman R. Combined immunomodulatory therapy in autoimmune autonomic ganglionopathy.
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8. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 6, 2010, No. 2 , , ,

Proposals for upgrading the postgraduate training programs "Neurosonology” and “Investigation of Autonomic Nervous System” were also made.
in carotid pathology, published in Issue 2/2008 of the Journal. It helps the experts to follow uniform rules for sonographic examinations and result interpretation, related to target groups, study frequency, location, type and severity of vascular pathology, follow up of the effect of treatment. Proposals for upgrading the postgraduate training programs "Neurosonology” and “Investigation of Autonomic Nervous System” were also made.
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9. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 7, 2011, No. 1 , , ,

According to the guideline for the diagnosis and management of syncope, version 2009 [4], syncope classification includes reflex syncope (with subgroups vasovagal, situational, carotid sinus syncope and atypical forms), syncope due to orthostatic hypotension (primary and secondary autonomic failure, drug-induced orthostatic hypotension and volume depletion) and cardiovascular syncope (rhythm-conduction disturbances or structural diseases).
Syncope is defined as a transient loss of consciousness due to transient global cerebral hypoperfusion characterized by rapid onset, short duration and spontaneous complete recovery [4]. According to the guideline for the diagnosis and management of syncope, version 2009 [4], syncope classification includes reflex syncope (with subgroups vasovagal, situational, carotid sinus syncope and atypical forms), syncope due to orthostatic hypotension (primary and secondary autonomic failure, drug-induced orthostatic hypotension and volume depletion) and cardiovascular syncope (rhythm-conduction disturbances or structural diseases). Syncope is common in the general population with occurrence of 18 to 40 per 1000 individuals [4]. Prognosis in patients with syncope varies considerably with etiology. Recurrences have a great impact on quality of life.
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such as primary and secondary autonomic dysfunction, cerebro-vascular diseases (“steal phenomenon” with subclavian artery stenosis and transitory ischemic attack in the vertebro-basilar system) and epilepsy.
such as primary and secondary autonomic dysfunction, cerebro-vascular diseases (“steal phenomenon” with subclavian artery stenosis and transitory ischemic attack in the vertebro-basilar system) and epilepsy.
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10. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 7, 2011, No. 2 , , ,

Autonomic neuropathy might induce increased blood shunting through skin arterio-venular anastomoses and to disturb nutritious capillary circulation [4, 11, 31].
Our study established increased values of skin perfusion in patients with diabetes mellitus. Skin microvascular blood flow in the limbs at normal temperature pass through arterio-venular shunts and by-passes nutritious capillary bed inducing capillary tissue ischemia [4, 11, 29]. Autonomic neuropathy might induce increased blood shunting through skin arterio-venular anastomoses and to disturb nutritious capillary circulation [4, 11, 31]. Early in the course of the disease microvascular perfusion at rest is increased [18, 29]. In condition of stress – tissue damage or a period of arterial occlusion restricted hyperemic response is observed [26, 28]. Increased skin microvascular perfusion is connected with poor glycemic control [13, 15, 34].
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Peripheral autonomic impairment in patients newly diagnosed with type II diabetes.
McDaid EA, Monaghan B, Parker AI, Hayes RY, Allen JA. Peripheral autonomic impairment in patients newly diagnosed with type II diabetes.
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11. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 8, 2012, No. 1 , , ,

Most often it is manifested as a symmetric sensorimotor and autonomic neuropathy [9].
Diabetic neuropathy is a late complication of diabetes observed in 50% of patients. Most often it is manifested as a symmetric sensorimotor and autonomic neuropathy [9]. Histological studies show neurogenic muscular atrophy with signs of chronic denervation and reinervation angular small muscle fibers, muscle fibers type “target”, grouping of muscle fibers in the form of bundle atrophy (Fig. 4).
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Primary autonomic failure:
Primary autonomic failure:
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pure autonomic failure, multiple system atrophy, Parkinson’s disease, Lewy body dementia.
pure autonomic failure, multiple system atrophy, Parkinson’s disease, Lewy body dementia.
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Secondary autonomic failure:
Secondary autonomic failure:
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In contrast to reflex syncope autonomic failure is associated with sympathetic afferent activity impairment resulting in inadequacy or absence of vasoconstriction [10].
In contrast to reflex syncope autonomic failure is associated with sympathetic afferent activity impairment resulting in inadequacy or absence of vasoconstriction [10]. Upon standing BP falls and presyncope or syncope occurs. Orthostatic hypotension is defined as a fall in BP during erect posture. We refer to classic orthostatic hypotension when a fall in systolic
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It could be present in patients with pure autonomic dysfunction, other forms of autonomic nervous system dysfunction and with volume depletion.
≥ 20 mmHg and diastolic BP ≥ 10 mmHg is observed during the first 3 min after standing [5]. It could be present in patients with pure autonomic dysfunction, other forms of autonomic nervous system dysfunction and with volume depletion. Initial orthostatic hypotension is characterized by a fall in BP > 40 mmHg immediately upon standing with spontaneous and rapid normalization of BP, making the period with hypotension and symptoms short (< 30 sec) [20]. Delayed (progressive) orthostatic hypotension is common among elderly patients and is observed between the 3rd and 30th min of erect positioning. It could be attributed to age-associated compensatory reflex dysfunction and to increased myocardial and arterial stiffness, which in turns increases the susceptibility to changes in preload [19]. Delayed orthostatic hypotension is characterized with slow and progressive decrease in BP after standing and the absence of reflex bradycardia (vagal reaction) differentiates it from reflex syncope.
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Role of autonomic reflexes in syncope associated with paroxysmal atrial fibrillation.
Brignole M, Gianfranchi L, Menozzi C, Raviele A, Oddone D, Lolli G, Bottoni N. Role of autonomic reflexes in syncope associated with paroxysmal atrial fibrillation.
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Consensus statement on the definition of orthostatic hypotension, pure autonomic failure, and multiple system atrophy.
Consensus statement on the definition of orthostatic hypotension, pure autonomic failure, and multiple system atrophy.
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In Th6 level injury or above there is a risk of life-threatening autonomic dysreflexia.
To increase the functional capacity of the spinal cord, various neurorehabilitation techniques are used [3]. In Th6 level injury or above there is a risk of life-threatening autonomic dysreflexia. Demineralization of bones is common in spinal cord injuries and appears very quickly after paralysis. Bone loss reaches 22% within three months. Pulmonary function is impaired in all patients with spinal cord traumas. Ultrasound imaging contributes to early detection of vascular complications [4].
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The Consensus Committee of the American Autonomic Society and the American Academy of Neurology.
The Consensus Committee of the American Autonomic Society and the American Academy of Neurology. Consensus statement on the definition of I orthostatic hypotension, pure autonomic failure, and multiple system atrophy.
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Consensus statement on the definition of I orthostatic hypotension, pure autonomic failure, and multiple system atrophy.
The Consensus Committee of the American Autonomic Society and the American Academy of Neurology. Consensus statement on the definition of I orthostatic hypotension, pure autonomic failure, and multiple system atrophy.
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12. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 2, 2012, No. 2 , , ,

Relative contribution of core and cutaneous temperatures to thermal comfort and autonomic responses in humans.
Frank S, Raja S, Bulcao C, Goldstein D. Relative contribution of core and cutaneous temperatures to thermal comfort and autonomic responses in humans.
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13. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 9, 2013, No. 1 , , ,

In 1983 she joined the Department for Diencephalic Pathology and Autonomic Nervous System Diseases at the 3rd Clinic of Neurology (St.
Ljudmila Zaprianova, a worthy woman, a dedicated doctor and an excellent teacher. She was borne on June 6, 1958 in Sofia. She graduated from the High Medical School in Sofia in 1982 with excellent grades. Her professional career began in the Hospital of the Ljubimez town. After winning a competitive examination for Assistant Professor at the Department of Neurology of the Medical University she worked successively at the Alexandrovska and the ISUL (Tzaritza Jovanna) Hospitals. In 1983 she joined the Department for Diencephalic Pathology and Autonomic Nervous System Diseases at the 3rd Clinic of Neurology (St. Naum Hospital) where she worked until her death.
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She was an author of a number of publications in the field of the autonomic physiology and pathology.
“Neurovegetative and Neuroendocrine Aspects of the Biological StressResponse in Patients with Hypothalamic Syndromes” and acquired the scientific degree “Doctor”. She was an author of a number of publications in the field of the autonomic physiology and pathology. During her work as a clinician she was approved as a competent and respected professional in the diagnostics and treatment of the diseases of autonomic nervous system and borderline endocrinology and psychiatry pathological conditions.
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During her work as a clinician she was approved as a competent and respected professional in the diagnostics and treatment of the diseases of autonomic nervous system and borderline endocrinology and psychiatry pathological conditions.
“Neurovegetative and Neuroendocrine Aspects of the Biological StressResponse in Patients with Hypothalamic Syndromes” and acquired the scientific degree “Doctor”. She was an author of a number of publications in the field of the autonomic physiology and pathology. During her work as a clinician she was approved as a competent and respected professional in the diagnostics and treatment of the diseases of autonomic nervous system and borderline endocrinology and psychiatry pathological conditions.
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14. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 9, 2013, No. 2 , , ,

In cases with pure autonomic failure the cerebral autoregulation seemed to be preserved if the MBP was maintained within the limit for brain autoregulation.
The pattern of orthostatic adjustment of the cerebral and systemic hemodynamics depended on the topic of the lesion, the type (“passive” or “active”) of the orthostatic challenge and the antigravity efficacy of the peripheral muscle pump. A paradoxical cerebral vasoconstriction due to hyperventilation was found in patients with postural tachycardia syndrome. In cases with pure autonomic failure the cerebral autoregulation seemed to be preserved if the MBP was maintained within the limit for brain autoregulation. During the induced neurally mediated syncope the selective loss of diastolic BFV and the increase in Pulsatility index were typically observed.
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with vascular and autonomic nervous system disorders.
with vascular and autonomic nervous system disorders.
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15. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 10, 2014, No. 1 , , ,

Autonomic dysbalance with increased sympathetic activity and/or parasympathetic depression, probably initiated by overstimulation of Pacinian corpuscles has been established [1, 9].
Many regulatory factors participate in pathophysiological mechanisms of vibrationinduced microvascular changes. Autonomic dysbalance with increased sympathetic activity and/or parasympathetic depression, probably initiated by overstimulation of Pacinian corpuscles has been established [1, 9]. Mechanical damage to blood vessels, vasoregulatory nerve elements, especially sympathetic vasoconstrictor nerves and receptors in fingers and digital cutaneous perivascular nerves containing the neuropeptide with powerful vasodilator properties calcitonin generelated peptide (CGRP) has been described [2, 3, 6, 10]. Pain-mediating nerve fibers and receptors, temperature nerve-endings, mechanoreceptors at the fingertips are affected by hand-arm vibration [5]. The neural deficit in digital skin of patients with VRP has a functional counterpart with reduced ability to propagate an axon-reflex vasodilator response [4]. Various degrees and forms of endothelial damage and dysfunctions may derive as a result of hand-arm vibration exposure [11].
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16. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 10, 2014, No. 2 , , ,

Primary investigation areas: cerebral vascular disease, cerebral hemodynamics, neurosonology and syndrome of orthostatic intolerance / autonomic nervous system; Participated in several investigation projects with other national and international centres, on the described areas, besides coordinating some clinical trials; Organized 93 courses and meetings, some of them international; Held 289 lectures and participated in 276 in scientific presentations.
Coordinator of the cerebrovascular investigation of the Unit of Cardiovascular Investigation of FMUP (since 2007). Primary investigation areas: cerebral vascular disease, cerebral hemodynamics, neurosonology and syndrome of orthostatic intolerance / autonomic nervous system; Participated in several investigation projects with other national and international centres, on the described areas, besides coordinating some clinical trials; Organized 93 courses and meetings, some of them international; Held 289 lectures and participated in 276 in scientific presentations. She is author of 197 papers published as abstracts and 55 published as full-texts in national and international journals, 5 chapters of books and edited 4 scientific publications and has received 6 scientific awards. She collaborates with various national and international work groups, within both clinical and investigation projects.
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cerebrovascular disease, neurosonology, cerebral hemodynamics, hemorheology, autonomic failure, orthostatic intolerance, gait motor control, neurorehabilitation.
cerebrovascular disease, neurosonology, cerebral hemodynamics, hemorheology, autonomic failure, orthostatic intolerance, gait motor control, neurorehabilitation.
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Human neuronal control of autonomic functional movements: interaction between central programs and afferent input.
Dietz V. Human neuronal control of autonomic functional movements: interaction between central programs and afferent input.
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17. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 11, 2015, No. 2 , , ,

autonomic nervous system, hand-arm vibration syndrome,
autonomic nervous system, hand-arm vibration syndrome,
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To measure heart rate variability and assess cardiac autonomic function in patients with hand-arm vibration syndrome and Raynaud's phenomenon (vRP).
To measure heart rate variability and assess cardiac autonomic function in patients with hand-arm vibration syndrome and Raynaud's phenomenon (vRP).
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The measurements of cardiac vagal and adrenergic autonomic responses by deep breathing established reduced heart rate variability in the patients with hand-arm vibration syndrome and scleroderma, while the LF power is higher in the patients with hand-arm vibration syndrome than in the control subjects.
Increased sympathetic and decreased parasympathetic modulation at rest in all patients with Raynaud's phenomenon was established. Abnormal heart rate variability with low power spectral density in the LF and HF components and reduction of the LF/HF ratio were obtained, pointing to reduced parasympathetic control in patients with scleroderma and Raynaud’s phenomenon. The measurements of cardiac vagal and adrenergic autonomic responses by deep breathing established reduced heart rate variability in the patients with hand-arm vibration syndrome and scleroderma, while the LF power is higher in the patients with hand-arm vibration syndrome than in the control subjects. The cardiac autonomic responses to orthostatism showed an increased sympathetic reactivity in the patients with hand-arm vibration syndrome and reduced reactivity in the patients with scleroderma. Hyperreactivity during orthostatism and cold test were established in the primary Raynaud’s phenomenon patients, confirming the increased activity of sympathetic nervous system.
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The cardiac autonomic responses to orthostatism showed an increased sympathetic reactivity in the patients with hand-arm vibration syndrome and reduced reactivity in the patients with scleroderma.
Increased sympathetic and decreased parasympathetic modulation at rest in all patients with Raynaud's phenomenon was established. Abnormal heart rate variability with low power spectral density in the LF and HF components and reduction of the LF/HF ratio were obtained, pointing to reduced parasympathetic control in patients with scleroderma and Raynaud’s phenomenon. The measurements of cardiac vagal and adrenergic autonomic responses by deep breathing established reduced heart rate variability in the patients with hand-arm vibration syndrome and scleroderma, while the LF power is higher in the patients with hand-arm vibration syndrome than in the control subjects. The cardiac autonomic responses to orthostatism showed an increased sympathetic reactivity in the patients with hand-arm vibration syndrome and reduced reactivity in the patients with scleroderma. Hyperreactivity during orthostatism and cold test were established in the primary Raynaud’s phenomenon patients, confirming the increased activity of sympathetic nervous system.
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The established cardiac autonomic dysregulation in patients with hand-arm vibration syndrome and Raynaud’s phenomenon could affect the clinical manifestations and the course of the disease.
The established cardiac autonomic dysregulation in patients with hand-arm vibration syndrome and Raynaud’s phenomenon could affect the clinical manifestations and the course of the disease.
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Spectral analysis of heart rate variability is a noninvasive method for quantitative evaluation of neural cardiovascular control and study of changes in the activity of the autonomic nervous system at rest and during functional effects.
Spectral analysis of heart rate variability is a noninvasive method for quantitative evaluation of neural cardiovascular control and study of changes in the activity of the autonomic nervous system at rest and during functional effects. The components of the heart rate variability assess the extent of the autonomic modulation and reflect distortions in baroreflex activity and autonomic dysfunction. Heart rate variability in short electrocardiographic recordings shows three typical peaks in the very low (0 to 0.04 Hz; VLF), low (0.04 to 0.15 Hz; LF) and respiratory or high (0.15 to 0.40 Hz; HF) frequency bands. Respiratory HF frequencies of R-R interval fluctuations reflect vagal efferent pathway to the heart, i.e. fluctuations in vagal activity. Low frequencies (LF) of the R-R intervals characterize sympathetic neural spectrum [11].
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The components of the heart rate variability assess the extent of the autonomic modulation and reflect distortions in baroreflex activity and autonomic dysfunction.
Spectral analysis of heart rate variability is a noninvasive method for quantitative evaluation of neural cardiovascular control and study of changes in the activity of the autonomic nervous system at rest and during functional effects. The components of the heart rate variability assess the extent of the autonomic modulation and reflect distortions in baroreflex activity and autonomic dysfunction. Heart rate variability in short electrocardiographic recordings shows three typical peaks in the very low (0 to 0.04 Hz; VLF), low (0.04 to 0.15 Hz; LF) and respiratory or high (0.15 to 0.40 Hz; HF) frequency bands. Respiratory HF frequencies of R-R interval fluctuations reflect vagal efferent pathway to the heart, i.e. fluctuations in vagal activity. Low frequencies (LF) of the R-R intervals characterize sympathetic neural spectrum [11]. Fluctuations in vagal and sympathetic neural activity at rest and under external influences vary over time and are in constant interaction.
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The two parts of the autonomic nervous system are controlled and balanced [3].
Heart rate variability in short electrocardiographic recordings shows three typical peaks in the very low (0 to 0.04 Hz; VLF), low (0.04 to 0.15 Hz; LF) and respiratory or high (0.15 to 0.40 Hz; HF) frequency bands. Respiratory HF frequencies of R-R interval fluctuations reflect vagal efferent pathway to the heart, i.e. fluctuations in vagal activity. Low frequencies (LF) of the R-R intervals characterize sympathetic neural spectrum [11]. Fluctuations in vagal and sympathetic neural activity at rest and under external influences vary over time and are in constant interaction. The two parts of the autonomic nervous system are controlled and balanced [3].
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Several studies show dysfunctions of the autonomic nervous system in hand-arm vibration syndrome (HAVS) besides the characteristic peripheral vascular, neurological and musculoskeletal disorders in the upper limbs [2, 5].
Several studies show dysfunctions of the autonomic nervous system in hand-arm vibration syndrome (HAVS) besides the characteristic peripheral vascular, neurological and musculoskeletal disorders in the upper limbs [2, 5]. Hyperreactivity of the sympathetic nervous system in hand-arm vibration syndorme patients during cold impact is established by elevated serum catecholamines [14].
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The aim of the study is to measure heart rate variability and assess cardiac autonomic function in patients with hand-arm vibration syndrome and Raynaud's phenomenon (vRP).
The aim of the study is to measure heart rate variability and assess cardiac autonomic function in patients with hand-arm vibration syndrome and Raynaud's phenomenon (vRP).
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The data about the ratio LF/HF of the spectral powers, which is a relative index of the balance between the sympathetic and parasympathetic autonomic nervous systems, did not show reliable differences in the values between the groups.
The data about the ratio LF/HF of the spectral powers, which is a relative index of the balance between the sympathetic and parasympathetic autonomic nervous systems, did not show reliable differences in the values between the groups.
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During orthostatic, deep breathing and cold tests the patients with scleroderma maintained similar heart rate variability, suggesting impaired baroreceptor and thermoregulatory modulation of the autonomic control and cardiovascular autonomic dysfunction.
compared to those in primary Raynaud's phenomenon, and healthy persons. During orthostatic, deep breathing and cold tests the patients with scleroderma maintained similar heart rate variability, suggesting impaired baroreceptor and thermoregulatory modulation of the autonomic control and cardiovascular autonomic dysfunction. The sympathetic activity was increased in the patients with scleroderma. A state of sympathetic arousal is suggested because of a reliable reduction of heart rate variability [16]. Reliable abnormalities in cardiovascular reflexes with sympathetic and parasympathetic dysfunctions suggestive of autonomic neuropathy in scleroderma have been described by other authors [7, 8, 13, 15].
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Reliable abnormalities in cardiovascular reflexes with sympathetic and parasympathetic dysfunctions suggestive of autonomic neuropathy in scleroderma have been described by other authors [7, 8, 13, 15].
compared to those in primary Raynaud's phenomenon, and healthy persons. During orthostatic, deep breathing and cold tests the patients with scleroderma maintained similar heart rate variability, suggesting impaired baroreceptor and thermoregulatory modulation of the autonomic control and cardiovascular autonomic dysfunction. The sympathetic activity was increased in the patients with scleroderma. A state of sympathetic arousal is suggested because of a reliable reduction of heart rate variability [16]. Reliable abnormalities in cardiovascular reflexes with sympathetic and parasympathetic dysfunctions suggestive of autonomic neuropathy in scleroderma have been described by other authors [7, 8, 13, 15].
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Hemodynamics in postural changes causes autonomic neural responses of the cardiovascular system, which buffers the fluctuations in blood pressure and causes baroreflex-mediated effects [12].
Hemodynamics in postural changes causes autonomic neural responses of the cardiovascular system, which buffers the fluctuations in blood pressure and causes baroreflex-mediated effects [12]. Cardiovascular autonomic functions in response to gravity-related baroreceptor stimulation showed enhanced sympathetic response in the first control and second vRP groups and suppressed reactivity in the fourth sclRP group. Hyperreactivity to orthostatic and cold tests was observed in pRP, confirming increased activity of the sympathetic nervous system. Patients with pRp have normal heart rate variability, but sympathetic hyperactivity to functional stimulation.
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Cardiovascular autonomic functions in response to gravity-related baroreceptor stimulation showed enhanced sympathetic response in the first control and second vRP groups and suppressed reactivity in the fourth sclRP group.
Hemodynamics in postural changes causes autonomic neural responses of the cardiovascular system, which buffers the fluctuations in blood pressure and causes baroreflex-mediated effects [12]. Cardiovascular autonomic functions in response to gravity-related baroreceptor stimulation showed enhanced sympathetic response in the first control and second vRP groups and suppressed reactivity in the fourth sclRP group. Hyperreactivity to orthostatic and cold tests was observed in pRP, confirming increased activity of the sympathetic nervous system. Patients with pRp have normal heart rate variability, but sympathetic hyperactivity to functional stimulation.
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The measurements of cardiac vagal and adrenergic autonomic responses by deep breathing established a reduced R-R variability in HAVS patients and scleroderma, but LF power was higher in the HAVS patients than in controls.
The study of R-R variability at rest and during deep breathing, determined by respiratory arrhythmia, reflects parasympathetic activity. The measurements of cardiac vagal and adrenergic autonomic responses by deep breathing established a reduced R-R variability in HAVS patients and scleroderma, but LF power was higher in the HAVS patients than in controls.
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Segmental vibration has an independent negative relation with indicators of heart variability and effects on autonomic functions [17].
Similar results in exposed to vibration have been established by other researchers [4]. Basal vagal activity according to the R-R variability is reduced in vibration disease [1, 5]. Studies suggest a prevalence of sympathetic tone in HAVS. Decreased parasympathetic activity was found in HAVS patients, which is a negative correlation with the duration of the vibration exposure [9, 10]. A number of authors found significant differences between the indices of heart rate variability during deep breathing and the duration of vibration exposure. Segmental vibration has an independent negative relation with indicators of heart variability and effects on autonomic functions [17]. Prolonged vibration exposure has a negative effect on the parasympathetic activity causing autonomic dysfunction [6].
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Prolonged vibration exposure has a negative effect on the parasympathetic activity causing autonomic dysfunction [6].
Basal vagal activity according to the R-R variability is reduced in vibration disease [1, 5]. Studies suggest a prevalence of sympathetic tone in HAVS. Decreased parasympathetic activity was found in HAVS patients, which is a negative correlation with the duration of the vibration exposure [9, 10]. A number of authors found significant differences between the indices of heart rate variability during deep breathing and the duration of vibration exposure. Segmental vibration has an independent negative relation with indicators of heart variability and effects on autonomic functions [17]. Prolonged vibration exposure has a negative effect on the parasympathetic activity causing autonomic dysfunction [6].
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and adrenergic autonomic responses by deep breathing established a reduced R-R variability in vibration disease, and scleroderma.
and adrenergic autonomic responses by deep breathing established a reduced R-R variability in vibration disease, and scleroderma. Cardiac autonomic orthostatic responses showed enhanced sympathetic reactivity in vibration disease and suppressed reactivity in scleroderma. Increased sympathetic activity was found in primary Raynaud’s phenomenon. Abnormal heart rate variability with low power spectral density at LF and HF components and reduced LF/HF ratio, reflecting reduced parasympathetic control was seen in scleroderma. The established cardiac autonomic dysregulation could affect the clinical manifestations and the course of the disease.
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Cardiac autonomic orthostatic responses showed enhanced sympathetic reactivity in vibration disease and suppressed reactivity in scleroderma.
and adrenergic autonomic responses by deep breathing established a reduced R-R variability in vibration disease, and scleroderma. Cardiac autonomic orthostatic responses showed enhanced sympathetic reactivity in vibration disease and suppressed reactivity in scleroderma. Increased sympathetic activity was found in primary Raynaud’s phenomenon. Abnormal heart rate variability with low power spectral density at LF and HF components and reduced LF/HF ratio, reflecting reduced parasympathetic control was seen in scleroderma. The established cardiac autonomic dysregulation could affect the clinical manifestations and the course of the disease.
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The established cardiac autonomic dysregulation could affect the clinical manifestations and the course of the disease.
and adrenergic autonomic responses by deep breathing established a reduced R-R variability in vibration disease, and scleroderma. Cardiac autonomic orthostatic responses showed enhanced sympathetic reactivity in vibration disease and suppressed reactivity in scleroderma. Increased sympathetic activity was found in primary Raynaud’s phenomenon. Abnormal heart rate variability with low power spectral density at LF and HF components and reduced LF/HF ratio, reflecting reduced parasympathetic control was seen in scleroderma. The established cardiac autonomic dysregulation could affect the clinical manifestations and the course of the disease.
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Autonomic stimulation and cardiovascular reflex activity in the hand-arm vibration syndrome.
Bovenzi M. Autonomic stimulation and cardiovascular reflex activity in the hand-arm vibration syndrome.
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Assessment of autonomic nervous function in patients with vibration syndrome using heart rate variation and plasma cyclic nucleotides.
Harada N, Kondo H, Kimura K. Assessment of autonomic nervous function in patients with vibration syndrome using heart rate variation and plasma cyclic nucleotides.
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Harada N Autonomic nervous function of hand-arm vibration syndrome patients.
Harada N Autonomic nervous function of hand-arm vibration syndrome patients.
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Heinonen E, Farkkila M, Forsstrom J, Antila K, Jalonen J, Korhonen O, Pyykko I Autonomic neuropathy and vibration exposure in forestry workers.
Heinonen E, Farkkila M, Forsstrom J, Antila K, Jalonen J, Korhonen O, Pyykko I Autonomic neuropathy and vibration exposure in forestry workers.
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Autonomic dysfunction in diffuse scleroderma vs CREST: an assessment by computerized heart rate variability.
Hermosillo AG, Ortiz R, Dabague J, Casanova JM, MartinezLavin M. Autonomic dysfunction in diffuse scleroderma vs CREST: an assessment by computerized heart rate variability.
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Autonomic neuropathy in systemic sclerosis.
Klimiuk PS, Taylor L, Baker RD, Jayson MI. Autonomic neuropathy in systemic sclerosis.
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Laskar MS, Harada N Assessment of autonomic nervous activity in hand-arm vibration syndrome patients using timeand frequency-domain analyses of heart rate variation.
Laskar MS, Harada N Assessment of autonomic nervous activity in hand-arm vibration syndrome patients using timeand frequency-domain analyses of heart rate variation.
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Pancera P, Sansone S, Presciuttini B, Montagna L, Ceru S, Lunardi C, Lechi A Autonomic nervous system dysfunction in sclerodermic and primary Raynaud's phenomenon.
Pancera P, Sansone S, Presciuttini B, Montagna L, Ceru S, Lunardi C, Lechi A Autonomic nervous system dysfunction in sclerodermic and primary Raynaud's phenomenon.
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Sato N, Kawamoto M, Yuge O, Suyama H, Sanuki M, Matsumoto C, Inoue K Surg Effects of pneumoperitoneum on cardiac autonomic nervous activity evaluated by heart rate variability analysis during sevoflurane, isoflurane, or propofol anesthesia.
Sato N, Kawamoto M, Yuge O, Suyama H, Sanuki M, Matsumoto C, Inoue K Surg Effects of pneumoperitoneum on cardiac autonomic nervous activity evaluated by heart rate variability analysis during sevoflurane, isoflurane, or propofol anesthesia.
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Yamamoto M, Yamasaki Y, Kodama M, Matsuhisa M, Kishimoto M, Ozaki H, Tani A, Ueda N, Iwasaki M, Hori M Impaired diurnal cardiac autonomic function in subjects with type 2 diabetes.
Yamamoto M, Yamasaki Y, Kodama M, Matsuhisa M, Kishimoto M, Ozaki H, Tani A, Ueda N, Iwasaki M, Hori M Impaired diurnal cardiac autonomic function in subjects with type 2 diabetes.
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A modern introduction to the study of the autonomic nervous system is presented.
The contemporary neurosonology is presented shortly, but maximally detailed and perfectly illustrated. Along with the latest innovations in the field of vascular pathology, neurosonology rises to a qualitatively new level certain areas of myology, neuroofthalmology, intraoperative navigation and even assists the diagnosis of neurodegenerative diseases. A modern introduction to the study of the autonomic nervous system is presented. In the near future the progress in this field will give us a new understanding and opportunities for the diagnosis and treatment of different somatic diseases. A substantive contribution is the section on the study and evaluation of gait – normal and pathological.
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P27 Cardiovascular Autonomic Dysfunction in Multiple Sclerosis.
P27 Cardiovascular Autonomic Dysfunction in Multiple Sclerosis.
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Autonomic dysfunction in multiple sclerosis (MS) is most often presented with urinary symptoms, while cardiovascular are less frequently reported.
Autonomic dysfunction in multiple sclerosis (MS) is most often presented with urinary symptoms, while cardiovascular are less frequently reported. They can be evaluated with cardiovascular autonomic tests.
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They can be evaluated with cardiovascular autonomic tests.
Autonomic dysfunction in multiple sclerosis (MS) is most often presented with urinary symptoms, while cardiovascular are less frequently reported. They can be evaluated with cardiovascular autonomic tests.
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To investigate the alterations in the cardiovascular autonomic function in patients with MS and to correlate them with the type, severity and duration of the disease.
To investigate the alterations in the cardiovascular autonomic function in patients with MS and to correlate them with the type, severity and duration of the disease.
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Non-invasive monitoring of heart rate, blood pressure and respiration at rest and during autonomic tests (metronomic breathing, head-up tilt and handgrip) was performed.
Non-invasive monitoring of heart rate, blood pressure and respiration at rest and during autonomic tests (metronomic breathing, head-up tilt and handgrip) was performed. The time domain and spectral analysis parameters of the heart rate variability were calculated. The autonomic examination was also applied in 57 age-matched healthy subjects.
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The autonomic examination was also applied in 57 age-matched healthy subjects.
Non-invasive monitoring of heart rate, blood pressure and respiration at rest and during autonomic tests (metronomic breathing, head-up tilt and handgrip) was performed. The time domain and spectral analysis parameters of the heart rate variability were calculated. The autonomic examination was also applied in 57 age-matched healthy subjects.
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Our findings reveal impairment of the autonomic function in patients with MS with predominating parasympathetic dysfunction.
Our findings reveal impairment of the autonomic function in patients with MS with predominating parasympathetic dysfunction. Correlation between the sympathetic autonomic dysfunction and the fatigue syndrome is also established.
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Correlation between the sympathetic autonomic dysfunction and the fatigue syndrome is also established.
Our findings reveal impairment of the autonomic function in patients with MS with predominating parasympathetic dysfunction. Correlation between the sympathetic autonomic dysfunction and the fatigue syndrome is also established.
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autonomic dysfunction, multiple sclerosis.
autonomic dysfunction, multiple sclerosis.
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18. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 13, 2017, No. 1 , , ,

Mixed Topics (pain and headache, neurorehabilitation, autonomic dysfunction).
Mixed Topics (pain and headache, neurorehabilitation, autonomic dysfunction).
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19. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 13, 2017, No. 2 , , ,

Тhe EAN course is aimed at providing very high level of scientific and practical knowledge in the fields of Interventional Vascular Neurology, advance of Nonvascular Neurosonology and interdisciplinary problems such as pain and headache, autonomic dysfunction and neurorehabilitation.
Тhe EAN course is aimed at providing very high level of scientific and practical knowledge in the fields of Interventional Vascular Neurology, advance of Nonvascular Neurosonology and interdisciplinary problems such as pain and headache, autonomic dysfunction and neurorehabilitation. Тhese topics of social importance will be presented by leading European experts.
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He specialized in Neurology, Clinical Neurophysiology, Neurological Intensive Care Medicine and Disorders of the Autonomic Nervous System (ANS), and holds German board certificates in Neurology and Psychiatry and in Psychotherapy.
Prof. Dr. med. Dr.med.habil. Max J. Hilz studied medicine at the Universities of Cologne and Erlangen-Nuremberg in Germany. After he had defended his doctoral thesis in 1980, he trained in Anesthesiology and Intensive Care Medicine and in Ear-Nose-and–Throat diseases (1980–1982). He started his residency in Neurology and Psychiatry at the University of Erlangen-Nuremberg. He specialized in Neurology, Clinical Neurophysiology, Neurological Intensive Care Medicine and Disorders of the Autonomic Nervous System (ANS), and holds German board certificates in Neurology and Psychiatry and in Psychotherapy. He also passed the board examination of the American Board of Electrodiagnostic Medicine. He is licensed to practice medicine in Germany, the United Kingdom, and in the State of New York, USA.
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Form September 1, 2016 to August 31, 2017, he was the Chair in Autonomic Neurology, and Director of the Clinical Department of Autonomic Neurology at the University College London, Institute of Neurology, Queen Square, London, UK.
Since June 2015, he is Professor of Neurology at the University of ErlangenNuremberg in Erlangen, Germany. He is also Adjunct Professor of Neurology at Icahn School of Medicine at Mount Sinai, New York, NY, USA. Form September 1, 2016 to August 31, 2017, he was the Chair in Autonomic Neurology, and Director of the Clinical Department of Autonomic Neurology at the University College London, Institute of Neurology, Queen Square, London, UK. He was Professor of Neurology in Medicine and Psychiatry at the New York University, New York, NY (1992–2013). He also served as the Associate Director of the NYU Dysautonomia Evaluation and Treatment Center (until 2007). He was deeply involved in clinical research regarding the pathophysiology of Familial Dysautonomia, also known as Riley-Day syndrome or Hereditary Sensory and Autonomic Neuropathy Type III, and in studies of Fabry disease that led to the approval of enzyme replacement therapy in the USA.
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He was deeply involved in clinical research regarding the pathophysiology of Familial Dysautonomia, also known as Riley-Day syndrome or Hereditary Sensory and Autonomic Neuropathy Type III, and in studies of Fabry disease that led to the approval of enzyme replacement therapy in the USA.
Since June 2015, he is Professor of Neurology at the University of ErlangenNuremberg in Erlangen, Germany. He is also Adjunct Professor of Neurology at Icahn School of Medicine at Mount Sinai, New York, NY, USA. Form September 1, 2016 to August 31, 2017, he was the Chair in Autonomic Neurology, and Director of the Clinical Department of Autonomic Neurology at the University College London, Institute of Neurology, Queen Square, London, UK. He was Professor of Neurology in Medicine and Psychiatry at the New York University, New York, NY (1992–2013). He also served as the Associate Director of the NYU Dysautonomia Evaluation and Treatment Center (until 2007). He was deeply involved in clinical research regarding the pathophysiology of Familial Dysautonomia, also known as Riley-Day syndrome or Hereditary Sensory and Autonomic Neuropathy Type III, and in studies of Fabry disease that led to the approval of enzyme replacement therapy in the USA.
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Prof. Hilz is experienced in the examination of small nerve fiber diseases and disorders of the autonomic nervous system, including hereditary sensory and autonomic neuropathies, diabetic neuropathies, and Fabry disease, and central autonomic disorders.
Prof. Hilz is experienced in the examination of small nerve fiber diseases and disorders of the autonomic nervous system, including hereditary sensory and autonomic neuropathies, diabetic neuropathies, and Fabry disease, and central autonomic disorders. He also served as an advisor to the European Medicines Agency, EMA, on issues related to autonomic nervous system dysfunction.
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He also served as an advisor to the European Medicines Agency, EMA, on issues related to autonomic nervous system dysfunction.
Prof. Hilz is experienced in the examination of small nerve fiber diseases and disorders of the autonomic nervous system, including hereditary sensory and autonomic neuropathies, diabetic neuropathies, and Fabry disease, and central autonomic disorders. He also served as an advisor to the European Medicines Agency, EMA, on issues related to autonomic nervous system dysfunction.
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Professor Hilz is a member of 16 national and international scientific societies and is on the board of several autonomic nervous system societies.
Professor Hilz is a member of 16 national and international scientific societies and is on the board of several autonomic nervous system societies. He currently co-chairs the Autonomic Nervous System Subspecialty Panel of the European Academy of Neurology, EAN. He also is PastPresident of the German Autonomic Society, Past-President of the European Federation of Autonomic Societies, and Past-Chair of the Autonomic Section of the American Academy of Neurology. He is ad hoc reviewer for more than 25 international scientific journals, a member of the editorial board of Clinical Autonomic Research, and Associate Clinical Editor of Autonomic Neuroscience: Basic and Clinical. He co-authored the guidelines of the German Neurological Society on syncope, the guidelines on erectile dysfunction and the guidelines of the German Diabetes Society on diabetic neuropathy. He has published more than 300 original and review articles in peer-reviewed journals and chapters in textbooks and presented his work at several hundred scientific conferences.
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He currently co-chairs the Autonomic Nervous System Subspecialty Panel of the European Academy of Neurology, EAN.
Professor Hilz is a member of 16 national and international scientific societies and is on the board of several autonomic nervous system societies. He currently co-chairs the Autonomic Nervous System Subspecialty Panel of the European Academy of Neurology, EAN. He also is PastPresident of the German Autonomic Society, Past-President of the European Federation of Autonomic Societies, and Past-Chair of the Autonomic Section of the American Academy of Neurology. He is ad hoc reviewer for more than 25 international scientific journals, a member of the editorial board of Clinical Autonomic Research, and Associate Clinical Editor of Autonomic Neuroscience: Basic and Clinical. He co-authored the guidelines of the German Neurological Society on syncope, the guidelines on erectile dysfunction and the guidelines of the German Diabetes Society on diabetic neuropathy. He has published more than 300 original and review articles in peer-reviewed journals and chapters in textbooks and presented his work at several hundred scientific conferences.
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He also is PastPresident of the German Autonomic Society, Past-President of the European Federation of Autonomic Societies, and Past-Chair of the Autonomic Section of the American Academy of Neurology.
Professor Hilz is a member of 16 national and international scientific societies and is on the board of several autonomic nervous system societies. He currently co-chairs the Autonomic Nervous System Subspecialty Panel of the European Academy of Neurology, EAN. He also is PastPresident of the German Autonomic Society, Past-President of the European Federation of Autonomic Societies, and Past-Chair of the Autonomic Section of the American Academy of Neurology. He is ad hoc reviewer for more than 25 international scientific journals, a member of the editorial board of Clinical Autonomic Research, and Associate Clinical Editor of Autonomic Neuroscience: Basic and Clinical. He co-authored the guidelines of the German Neurological Society on syncope, the guidelines on erectile dysfunction and the guidelines of the German Diabetes Society on diabetic neuropathy. He has published more than 300 original and review articles in peer-reviewed journals and chapters in textbooks and presented his work at several hundred scientific conferences.
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He is ad hoc reviewer for more than 25 international scientific journals, a member of the editorial board of Clinical Autonomic Research, and Associate Clinical Editor of Autonomic Neuroscience: Basic and Clinical.
Professor Hilz is a member of 16 national and international scientific societies and is on the board of several autonomic nervous system societies. He currently co-chairs the Autonomic Nervous System Subspecialty Panel of the European Academy of Neurology, EAN. He also is PastPresident of the German Autonomic Society, Past-President of the European Federation of Autonomic Societies, and Past-Chair of the Autonomic Section of the American Academy of Neurology. He is ad hoc reviewer for more than 25 international scientific journals, a member of the editorial board of Clinical Autonomic Research, and Associate Clinical Editor of Autonomic Neuroscience: Basic and Clinical. He co-authored the guidelines of the German Neurological Society on syncope, the guidelines on erectile dysfunction and the guidelines of the German Diabetes Society on diabetic neuropathy. He has published more than 300 original and review articles in peer-reviewed journals and chapters in textbooks and presented his work at several hundred scientific conferences.
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cerebrovascular disease, neurosonology, cerebral hemodynamics, hemorheology, autonomic failure, orthostatic intolerance, gait motor control, neurorehabilitation.
cerebrovascular disease, neurosonology, cerebral hemodynamics, hemorheology, autonomic failure, orthostatic intolerance, gait motor control, neurorehabilitation.
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It is a common late complication of diabetes, often manifested as a symmetric sensorimotor and autonomic neuropathy.
It is a common late complication of diabetes, often manifested as a symmetric sensorimotor and autonomic neuropathy. Histological studies show neurogenic muscular atrophy with signs of chronic denervation and reinervation – angular small muscle fibers, muscle fibers type “target”, grouping of muscle fibers in the form of bundle atrophy. The neurogenic damage of calf muscles are proved by different methods – clinical, neurophysiologic, neuroimaging, etc. [17]. The simultaneous usage of EMG and myosonography helps for evaluation of the severity of peripheral nerves damage and the changes in cross-striated muscles that could contribute to distinguish primary from secondary myogenic lesions in peripheral neuropathy [8]. Myosonographic patterns in low extremity neuropathy demonstrate bundle atrophy with mild to severe involvement of both lateral heads of triceps surae muscle, which correlates with the
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This theory explains the fact that resulting in reduced central pain suppression in BMS individuals; 4) Disturbances in the autonomic innervation and oral blood flow;
Centrally mediated alteration in the modulation of nociceptive processing. This theory explains the fact that resulting in reduced central pain suppression in BMS individuals; 4) Disturbances in the autonomic innervation and oral blood flow;
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subjects the normal autonomic response provides adaptive changes in the systemic hemodynamics that keep the cerebral circulation stable and prevent from orthostatic hypotension [3, 25].
subjects the normal autonomic response provides adaptive changes in the systemic hemodynamics that keep the cerebral circulation stable and prevent from orthostatic hypotension [3, 25].
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Autonomic control of heart rate during exercise studies by heart rate variability spectral analisis.
Yamamoto Y, Hughson R, Peterson J. Autonomic control of heart rate during exercise studies by heart rate variability spectral analisis.
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Neurally mediated syncope and syncope due to autonomic failure: differences and similarities.
Kaufmann H. Neurally mediated syncope and syncope due to autonomic failure: differences and similarities.
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20. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 14, 2018, No. 1 , , ,

Starting with Introduction it is very well structured and organized in twelve chapters: Reflexes, Sensation, Motor Activity, Cranial Nerves & Specialized Sensation, Peripheral Nervous System, Autonomic Nervous System, Higher Cortical Functions, Cortical Syndromes, General
Kamelia Genova, Dr. Radostina Dimova, Prof. Daniela Lubenova, Ass. Prof. Pane Popov and Assoc. Prof. Boyko Stamenov, who gave their excellent contributions. Starting with Introduction it is very well structured and organized in twelve chapters: Reflexes, Sensation, Motor Activity, Cranial Nerves & Specialized Sensation, Peripheral Nervous System, Autonomic Nervous System, Higher Cortical Functions, Cortical Syndromes, General
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The EAN course successfully targeted its aim at providing a high level of scientific and practical knowledge in the fields of Interventional Vascular Neurology, advance of Nonvascular Neurosonology and interdisciplinary problems such as pain and headache, autonomic dysfunction and neurorehabilitation.
The EAN course successfully targeted its aim at providing a high level of scientific and practical knowledge in the fields of Interventional Vascular Neurology, advance of Nonvascular Neurosonology and interdisciplinary problems such as pain and headache, autonomic dysfunction and neurorehabilitation. These topics of social importance were presented by leading European experts.
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21. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 14, 2018, No. 2 , , ,

Haemodynamics in postural change causes autonomic neural responses to the cardiovascular system leading to changes in blood pressure and causing a barroreflective-mediated effect.
Haemodynamics in postural change causes autonomic neural responses to the cardiovascular system leading to changes in blood pressure and causing a barroreflective-mediated effect. Orthostatic autoregulation is an adaptive and compensatory mechanism against the gravitational redistribution of blood during the transition from horizontal to upright body position. Physical activity causes changes in the cerebral blood flow, which depends on their type, intensity and duration.
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The Bulgarian version of the SCOPAAUT questionnaire (SCOPA-AUT-BG) was used for assessment of cardiovascular autonomic symptoms.
Fifty-five PD patients (32 males and 23 females) at mean age 64.5±8.9 years and 40 age-matched healthy controls were included in the study. The Bulgarian version of the SCOPAAUT questionnaire (SCOPA-AUT-BG) was used for assessment of cardiovascular autonomic symptoms. All participants also underwent a head-up tilt test at 60 degrees for 10 minutes.
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Her research fields are focused on cerebrovascular diseases, neurosonology, hemorheology, autonomic failure, gait motor control, and neurorehabilitation.
Acad. Titianova has specialized in Neurology, Neurosonology, Neurorehabilitation and gait motor control at prestigious universities in Austria, USA, Mexico, Germany and Finland. Her publishing activity includes monographs, textbooks, manuals, and over 250 scientific papers, many of them internationally sited. She is a co-author of national and international teaching guides, recent editions of Cambridge University Press (UK) and Di Libros Editora LTDA (Brazil). Her research fields are focused on cerebrovascular diseases, neurosonology, hemorheology, autonomic failure, gait motor control, and neurorehabilitation. She is recognized as an international expert in Neurosology; introduced, developed and validated
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22. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 15, 2019, No. 1 , , ,

The Influence of Gender on Cardiovascular Autonomic Function in Patients with Parkinson’s Disease
The Influence of Gender on Cardiovascular Autonomic Function in Patients with Parkinson’s Disease
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The Influence of Gender on Cardiovascular Autonomic Function in Patients with Parkinson's Disease
The Influence of Gender on Cardiovascular Autonomic Function in Patients with Parkinson's Disease
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autonomic functions, cardiovascular function,
autonomic functions, cardiovascular function,
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To assess the influence of gender on the cardiovascular autonomic function in patients with Parkinson's disease.
To assess the influence of gender on the cardiovascular autonomic function in patients with Parkinson's disease.
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For evaluation of the cardiovascular autonomic symptoms, a SCOPA AUT-BG questionnaire was used.
Fifty-five patients with Parkinson's disease (32 men and 23 women) of average age 64.5±8.9 years and 40 healthy controls were examined. For evaluation of the cardiovascular autonomic symptoms, a SCOPA AUT-BG questionnaire was used. Also a non-invasive assessment of heart rate variability at rest and during head-up tilt (HUT) was performed and the timeand frequencydomain heart rate variability parameters were determined.
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The average scores of the cardiovascular and thermoregulatory autonomic subscales were significantly higher in female patients with Parkinson's disease (p>0.05).
The average scores of the cardiovascular and thermoregulatory autonomic subscales were significantly higher in female patients with Parkinson's disease (p>0.05). No significant gender differences in time and frequency heart rate variability parameters at rest in the two investigated groups were found. In healthy controls and in male Parkinson's disease patients head-up tilt provoked sympathetic activation with significant decrease in the duration of the RR interval. In contrast, the test did not change the duration of the RR interval in women.
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The established gender differences in cardiovascular autonomic function in patients with Parkinson's disease suggest a gender dependent expression and their diagnosis allows better clinical evaluation, treatment and prognosis of the disease.
The established gender differences in cardiovascular autonomic function in patients with Parkinson's disease suggest a gender dependent expression and their diagnosis allows better clinical evaluation, treatment and prognosis of the disease.
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Influence of Gender on Cardiovascular Autonomic Function in Patients with Parkinson's Disease
Influence of Gender on Cardiovascular Autonomic Function in Patients with Parkinson's Disease
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Cardiovascular autonomic disorders are part of the non-motor symptoms of the disease and have an impact on the quality of life, disability, and mortality of patients with PD [4, 21, 26, 28].
The importance of gender for non-motor symptoms in PD has been poorly studied; the interest for this relationship has increased over the last decade [15, 18, 24, 30, 32]. Cardiovascular autonomic disorders are part of the non-motor symptoms of the disease and have an impact on the quality of life, disability, and mortality of patients with PD [4, 21, 26, 28].
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Having in mind the existing gender dysmorphism in the dopaminergic system in the basal human ganglia [8, 11, 29], as well as the ambiguous intergender differences and correlations in the myocardial cardiac innervation in MIBG scintigraphy in patients with PD [10, 31, 33], a question arises whether gender has an effect on cardiovascular autonomic dysfunction in these patients.
Over the last 20 years, it has been shown that, in addition to the nigrostriatal system, loss of catecholaminergic neurons is also seen in the heart leading to cardiac dysautonomia [9]. Having in mind the existing gender dysmorphism in the dopaminergic system in the basal human ganglia [8, 11, 29], as well as the ambiguous intergender differences and correlations in the myocardial cardiac innervation in MIBG scintigraphy in patients with PD [10, 31, 33], a question arises whether gender has an effect on cardiovascular autonomic dysfunction in these patients. Despite the increasing number of studies related to this type of non-motor PD manifestations, the intergender differences still remain not enough studied.
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The purpose of this study is to evaluate the gender impact on cardiovascular autonomic function in patients with PD.
The purpose of this study is to evaluate the gender impact on cardiovascular autonomic function in patients with PD.
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The inclusion criteria were: clinical PD diagnosis according to the criteria of the UK Parkinson's disease Society, Brain Bank [13], lack of cognitive impairment as assessed by the Mini-Mental State Examination (MMSE), no history of chronic somatic or other diseases affecting primarily or secondarily the autonomic nervous system and administration of antiparkinsonian therapy alone.
The study included 55 patients with PD and 40 agerelated healthy individuals. The inclusion criteria were: clinical PD diagnosis according to the criteria of the UK Parkinson's disease Society, Brain Bank [13], lack of cognitive impairment as assessed by the Mini-Mental State Examination (MMSE), no history of chronic somatic or other diseases affecting primarily or secondarily the autonomic nervous system and administration of antiparkinsonian therapy alone. Most of the patients (76%) were on levodopa therapy, with an average daily dose 522.2±379.1 mg/day.
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A validated in Bulgarian questionnaire for evaluating the autonomic dysfunctions of Parkinson's disease (SCOPA-AUT-BG) [17] was applied.
A validated in Bulgarian questionnaire for evaluating the autonomic dysfunctions of Parkinson's disease (SCOPA-AUT-BG) [17] was applied. The internal consistency of SCOPA-AUT-BG was good (Cronbach’s
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The evaluation subscale for the cardiac autonomic functions contains 3 questions.
=0.79) and showed psychometric properties equal to the original version of the scale. It contains 25 questions, grouped in 6 subclasses, related to clinical manifestations belonging to different functional systems: gastrointestinal, urinary, cardiovascular, thermoregulatory, pupillary and sexual. The evaluation subscale for the cardiac autonomic functions contains 3 questions.
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In addition to the questionnaire, the cardiovascular autonomic function was also evaluated by short-term heart rate variability monitoring (HRV).
In addition to the questionnaire, the cardiovascular autonomic function was also evaluated by short-term heart rate variability monitoring (HRV). The study was conducted in a specialized laboratory at 23°-24°C without visual and/or acoustic stimuli. Heart rate and breathing were monitored in lying position (10 minutes) and during 5-minute head-up tilt at sixty degrees. The R-R intervals were registered by a MP100 computerized system with an ECG module (Biopac system Inc., USA). The time parameters: mean R-R interval (ms), mean heart rate (bpm), mean standard deviation of the R-R interval (SDNN) and, low-frequency (LF, 0,04-0,15 Hz), high-frequency (HF 0,15-0,40 Hz) spectral characteristics and their ratio (LF/HF) were calculated. In order to eliminate the interindividual (intragroup) differences in the spectral characteristics, the relative spectral characteristics were calculated by applying standardization of the low – and high-frequency spectral components as follows: LF%=LF/ (LF+HF)*100 and HF%=HF/((LF+HF)*100.
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Influence of Gender on Cardiovascular Autonomic Function in Patients with Parkinson's Disease
Influence of Gender on Cardiovascular Autonomic Function in Patients with Parkinson's Disease
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According to the data from the applied questionnaire for autonomic symptoms (SCOPAAUT-BG), the total score of autonomic dysfunction in Parkinson's disease patients was significantly higher than that of the control group.
According to the data from the applied questionnaire for autonomic symptoms (SCOPAAUT-BG), the total score of autonomic dysfunction in Parkinson's disease patients was significantly higher than that of the control group. A significant difference (Mann-Whitney U test, p
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Influence of Gender on Cardiovascular Autonomic Function in Patients with Parkinson's Disease
Influence of Gender on Cardiovascular Autonomic Function in Patients with Parkinson's Disease
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[23] that healthy older women over 60 have a more preserved cardiac autonomic modulation compared to men.
With age (after 50 years of age), the gender differences in the parasympathetic functions disappear, while the sympathetic dominance in men decreases only after the age of 60 [14]. Considering the age of the examined healthy individuals we assume that the lack of intergender differences in regard to the HRV is most probably related to their older age. Applying the head-up tilt test to the control group leads to the expected sympathetic activation. A shortening of the R-R interval and an increase of the LF/HF ratio due to a rise of LF and a decrease in HF was observed. However, this reactivity is more pronounced in women, which is in agreement with Persiguini at al. [23] that healthy older women over 60 have a more preserved cardiac autonomic modulation compared to men.
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Taking into account the age at which the clinical manifestation of the disease begins (in the case of reduced HRV at rest), it is very likely that the influence of gender on the cardiovascular autonomic function in PD patients will only occur in functional autonomic tests.
Unlike healthy individuals, patients with PD have a reduced sympathetic activation. At rest, no intergender differences are found, whereas the application of the orthostatic test shows an interaction between gender and provocation. Taking into account the age at which the clinical manifestation of the disease begins (in the case of reduced HRV at rest), it is very likely that the influence of gender on the cardiovascular autonomic function in PD patients will only occur in functional autonomic tests. The preserved shortening of the R-R interval on orthostatic test in men with PD (similar to healthy controls) could be due to the fact that gender-related differences in parasympathetic function disappear after the age of 50, while sympathetic dominance in men disappears significantly later [14], e.g. the age influence could not be excluded.
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The intergender differences we have identified in the questionnaire and in the HRV analysis in PD patients give us the reason to assume that the cardiac autonomic disorders could be genderdependent.
The intergender differences we have identified in the questionnaire and in the HRV analysis in PD patients give us the reason to assume that the cardiac autonomic disorders could be genderdependent. We suppose that taking into account the age changes occurring in the autonomic nervous system, it is likely that the gender differences could occur only after provocation, (both in the everyday life of the patients, after a change in the position of the body and during specialized autonomic tests).
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We suppose that taking into account the age changes occurring in the autonomic nervous system, it is likely that the gender differences could occur only after provocation, (both in the everyday life of the patients, after a change in the position of the body and during specialized autonomic tests).
The intergender differences we have identified in the questionnaire and in the HRV analysis in PD patients give us the reason to assume that the cardiac autonomic disorders could be genderdependent. We suppose that taking into account the age changes occurring in the autonomic nervous system, it is likely that the gender differences could occur only after provocation, (both in the everyday life of the patients, after a change in the position of the body and during specialized autonomic tests).
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The application of a set of tools (selfassessment questionary and specialized functional autonomic tests) to identify the intergender differences in the different aspects of the Parkinson’s disease, motor and nonmotor, gives opportunity for more precise clinical evaluation, therapy, and prognosis of the disease.
The application of a set of tools (selfassessment questionary and specialized functional autonomic tests) to identify the intergender differences in the different aspects of the Parkinson’s disease, motor and nonmotor, gives opportunity for more precise clinical evaluation, therapy, and prognosis of the disease.
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Influence of Gender on Cardiovascular Autonomic Function in Patients with Parkinson's Disease
Influence of Gender on Cardiovascular Autonomic Function in Patients with Parkinson's Disease
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Influence of age and gender on autonomic regulation of heart.
Abhishekh HA, Nisarga P, Kisan R, Meghana A, Chandran S, TrichurRaju, Sathyaprabha TN. Influence of age and gender on autonomic regulation of heart.
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Effects of gender and aging on differential autonomic responses to orthostatic maneuvers.
Barantke M, Krauss T, Ortak J, Lieb W, Reppel M, Burgdorf C, Pramstaller PP, Schunkert H, Bonnemeier H. Effects of gender and aging on differential autonomic responses to orthostatic maneuvers.
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Early abnormalities of vascular and cardiac autonomic control in Parkinson’s disease without orthostatic hypotension.
Barbic F, Perego F, Canesi M, Gianni M, Biagiotti S, Constantino G, Pezzoli G, Porta A, Malliani a, Furlan F. Early abnormalities of vascular and cardiac autonomic control in Parkinson’s disease without orthostatic hypotension.
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Effects of age and gender on autonomic control of blood pressure dynamics.
Barnett SR, Morin RJ, Kiely DK, Gagnon M, Azhar G, Knight EL, Nelson JC, Lipsitz LA. Effects of age and gender on autonomic control of blood pressure dynamics.
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Is reduced myocardial sympathetic innervation associated with clinical symptoms of autonomic impairment in idiopathic Parkinson'sdisease?
bender K, Kirsch CM, Hellwig D, Spiegel J. Is reduced myocardial sympathetic innervation associated with clinical symptoms of autonomic impairment in idiopathic Parkinson'sdisease?
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Validation of the Bulgarian version of Scales for Outcomes in Parkinson's Disease Autonomic (SCOPA-AUT-BG).
Mantarova S, Velcheva I, Georgieva S, Stambolieva K. Validation of the Bulgarian version of Scales for Outcomes in Parkinson's Disease Autonomic (SCOPA-AUT-BG).
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Gender differences in agerelated changes in cardiac autonomic nervous function.
Moodithaya S, Avadhany ST. Gender differences in agerelated changes in cardiac autonomic nervous function.
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Spectral and symbolic analysis of the effect of gender and postural change on cardiac autonomic modulation in healthy elderly subjects.
Perseguini NM, Takahashi AC, Rebelatto JR, Silva E, BorghiSilva A, Porta A, Montano N, Catai AM. Spectral and symbolic analysis of the effect of gender and postural change on cardiac autonomic modulation in healthy elderly subjects.
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autonomic dysfunction in Parkinsondisease.
autonomic dysfunction in Parkinsondisease.
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Patient-reported autonomic symptoms in Parkinson disease.
Verbaan D, Marinus J, Visser M, van Rooden SM, Stiggelbout AM, van Hilten JJ. Patient-reported autonomic symptoms in Parkinson disease.
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Keywords related to: 'autonomic'

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