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20 results of 7 texts with exact phrase : 'cerebral vasomotor reactivity'.

1. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, Vol. 1, 2005 , , ,

Pravastatin improves cerebral vasomotor reactivity in patients with subcortical small-vessel disease.
Sterzer P, Meintzschel F, Rosler A, Lanfermann H, Steinmetz H, Sitzer M. Pravastatin improves cerebral vasomotor reactivity in patients with subcortical small-vessel disease.
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2. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 2, 2006, No. 2 , , ,

Thus, the differences between CBF at rest and after the induction of hypercapnia reflect the state of cerebral vasomotor reactivity (VMR) and, hence cerebrovascular reserve capacity.
) on the cerebral circulation are mostly demonstrated in resistance brain arterioles and play an important role in cerebral autoregulation that enables relatively constant cerebral blood flow (CBF) during variations of cerebral perfusion pressure. Thus, the differences between CBF at rest and after the induction of hypercapnia reflect the state of cerebral vasomotor reactivity (VMR) and, hence cerebrovascular reserve capacity. VMR is defined as the vasodilation capacity of cerebral arterioles to external stimuli, such as increasing extracellular pCO
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cerebral vasomotor reactivity by various vasodilating stimuli: comparison of CO
cerebral vasomotor reactivity by various vasodilating stimuli: comparison of CO
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3. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 9, 2013, No. 1 , , ,

Cerebral vasomotor reactivity
Cerebral vasomotor reactivity
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4. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 9, 2013, No. 2 , , ,

Cerebral Vasomotor Reactivity in Patients with Acute Ischemic Stroke.
Cerebral Vasomotor Reactivity in Patients with Acute Ischemic Stroke.
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CEREBRAL VASOMOTOR REACTIVITY IN PATIENTS WITH ACUTE ISCHEMIC STROKE
CEREBRAL VASOMOTOR REACTIVITY IN PATIENTS WITH ACUTE ISCHEMIC STROKE
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5. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 10, 2014, No. 2 , , ,

Cerebral Vasomotor Reactivity in Clinical Settingsa
Cerebral Vasomotor Reactivity in Clinical Settingsa
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Cerebral Vasomotor Reactivity in Clinical Settings.
Cerebral Vasomotor Reactivity in Clinical Settings.
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Cerebrovascular Disorders (genetics, path-physiology of arteriosclerosisespecially the role of insulin resistance in atherogenesis, asymptomatic carotid artery stenosis and vascular dementia, rare causes of stroke especially in young adults), Ultrasound Techniques in Neurology (Power Triplex Color Doppler, Transcranial Doppler, detection of the circulating micro emboli and cerebral vasomotor reactivity testing, sonothrombolysis), chronic headaches (co-morbidity of migraine, chronic tension type of headache, rare headaches-SUNCT, cluster headache, paroxysmal hemicranias), Neuropsychology and Dementia, Movement Disorders (neuroimaging techniques, brain parenchyma sonography).
Cerebrovascular Disorders (genetics, path-physiology of arteriosclerosisespecially the role of insulin resistance in atherogenesis, asymptomatic carotid artery stenosis and vascular dementia, rare causes of stroke especially in young adults), Ultrasound Techniques in Neurology (Power Triplex Color Doppler, Transcranial Doppler, detection of the circulating micro emboli and cerebral vasomotor reactivity testing, sonothrombolysis), chronic headaches (co-morbidity of migraine, chronic tension type of headache, rare headaches-SUNCT, cluster headache, paroxysmal hemicranias), Neuropsychology and Dementia, Movement Disorders (neuroimaging techniques, brain parenchyma sonography).
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Cerebral Vasomotor Reactivity in Clinical Settings
Cerebral Vasomotor Reactivity in Clinical Settings
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carotid stenosis, cerebral vasomotor reactivity, transcranial Doppler sonography
carotid stenosis, cerebral vasomotor reactivity, transcranial Doppler sonography
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The cerebral vasomotor reactivity (VMR) indicates the ability of the cerebral arterioles to change their vascular tone under external stimuli.
The cerebral vasomotor reactivity (VMR) indicates the ability of the cerebral arterioles to change their vascular tone under external stimuli. Greatest influence on the VMR exert age, endothelial functions and blood rheological properties. The most frequent influences are inhalatory induced changes in the partial pressure of carbon dioxide and infusion of acetazolamide. The alterations in the cerebral circulation are examined mainly with transcranial Doppler sonography or magnetic resonance imaging. In patients with carotid stenoses and cerebral infarctions and also in other diseases the estimation of the VMR is important for evaluating the pathogenetic mechanisms and the clinical outcome and for selecting the therapeutic behavior in these patients.
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The cerebral vasomotor reactivity (VMR) indicates the ability of the cerebral arterioles to change their vascular tone under external stimuli.
The cerebral vasomotor reactivity (VMR) indicates the ability of the cerebral arterioles to change their vascular tone under external stimuli. The hemodynamic response after applying stimuli for assessment of the VMR is mainly related to metabolic mechanisms. The integrity of the vascular endothelium [22] and the influence of some endothelial factors, especially of NO are important for the regulation of the VMR. The tone of the cerebral arterioles is particularly sensitive to changes in the carbon dioxide partial pressure (PaCO
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Metabolic syndrome and cerebral vasomotor reactivity.
Giannopoulos S, Boden-Albalaa B, Choia JH, Carreraa E, Doylea M, Pereza T, Marshall RS. Metabolic syndrome and cerebral vasomotor reactivity.
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Improved Cerebral Vasomotor Reactivity After Exercise Training in Hemiparetic Stroke Survivors.
Ivey FM, Ryan AS, Hafer-Macko CE, Macko RF. Improved Cerebral Vasomotor Reactivity After Exercise Training in Hemiparetic Stroke Survivors.
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Cerebral vasomotor reactivity and risk of mortality: the Rotterdam Study.
Portegies ML, de Bruijn RF, Hofman A, Koudstaal PJ, Ikram MA. Cerebral vasomotor reactivity and risk of mortality: the Rotterdam Study.
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6. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 11, 2015, No. 1 , , ,

At the end of the lecture session, Professor Irena Velcheva spoke about Cerebral vasomotor reactivity in clinical settings.
Professor Claudio Baracchini (Italy) presented his research “Ultrasound study of intracranial stenoses: preand postendovascular treatment”. Professor Massimo Del Sette (Italy) gave wonderful presentation about Sonothrombolysis. Professor Milija Mijajlovic (Serbia) described “Ultrasound imaging of brain parenchyma, temporal arteries and orbita”. At the end of the lecture session, Professor Irena Velcheva spoke about Cerebral vasomotor reactivity in clinical settings.
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7. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 14, 2018, No. 2 , , ,

A preliminary study already in 1996 suggested that impaired cerebral vasomotor reactivity assessed after acetazolamide administration predicted ischemic cerebral events [7].
10 min. Usually the effect of acetazolamide is monitored by detection of the pulsatility index and middle cerebral artery mean flow velocity for 20–30 min. The advantage of acetazolamide is that it does not influence the basic cardiovascular parameters [19]. A preliminary study already in 1996 suggested that impaired cerebral vasomotor reactivity assessed after acetazolamide administration predicted ischemic cerebral events [7].
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Is impaired cerebral vasomotor reactivity a predictive factor of stroke in asymptomatic patients?
Gur AY, Bova I, Bornstein NM. Is impaired cerebral vasomotor reactivity a predictive factor of stroke in asymptomatic patients?
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The positive or negative effect of different pharmacotherapies (statins, antihypertensives, antiparkinson drugs, NSAIDS, diuretics, etc.) on cerebral vasomotor reactivity could be estimated.
The impairment of dilative capacity of brain arterioles can be also measured noninvasively, without radiation hazard, by transcranial Doppler. The positive or negative effect of different pharmacotherapies (statins, antihypertensives, antiparkinson drugs, NSAIDS, diuretics, etc.) on cerebral vasomotor reactivity could be estimated.
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