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Also try: venous sinus thrombosis, thrombosis of dural sinuses, cerebral venous sinus thrombosis,

38 results of 9 texts with exact phrase : 'sinus thrombosis'.

1. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 2, 2006, No. 2 , , ,

gunshot injury, preudotumour cerebri, venous sinus thrombosis
gunshot injury, preudotumour cerebri, venous sinus thrombosis
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Good final results of treatment, without any changes in sinus thrombosis is due to rapid compensation of venous blood flow by anastomoses, “intracranial decompression” by CSF and cerebral detritus leakage, surgical and therapeutical control of the brain edema, despite of lack of effect of anticoagulant therapy in one of the patients.
Good final results of treatment, without any changes in sinus thrombosis is due to rapid compensation of venous blood flow by anastomoses, “intracranial decompression” by CSF and cerebral detritus leakage, surgical and therapeutical control of the brain edema, despite of lack of effect of anticoagulant therapy in one of the patients.
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Heparin Urokinasae treatment in aseptic dural sinus thrombosis.
Di Rocco C, Janne Lli A, Leone G, Moschini M, Valori VM. Heparin Urokinasae treatment in aseptic dural sinus thrombosis.
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Superior sagittal sinus thrombosis.
Gttelfinger DM, Kokemen E. Superior sagittal sinus thrombosis.
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Hanley DF, Feldman E, Borel CO, Rosenbaum AE, Goldberg AL.Treatment of sagittal sinus thrombosis associated with cerebral hemorrhage and intracranial hypertension.
Hanley DF, Feldman E, Borel CO, Rosenbaum AE, Goldberg AL.Treatment of sagittal sinus thrombosis associated with cerebral hemorrhage and intracranial hypertension.
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2. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 3, 2007, No. 2 , , ,

Further recommendations are provided for the prevention of recurrent stroke in a variety of other specific circumstances, including arterial dissections, patent foramen ovale, hyperhomocysteinemia, hypercoagulable states, sickle cell disease, cerebral venous sinus thrombosis, stroke among women, particularly with regard to pregnancy and the use of postmenopausal hormones, the use of anticoagulation after cerebral hemorrhage, and special approaches for the implementation of guidelines and their use in high-risk populations. [
A comprehensive review of the published guidelines and recommendations on the prevention of ischemic stroke among survivors of ischemic stroke or transient ischemic attack is presented. Evidence-based recommendations are included for the control of risk factors, interventional approaches for atherosclerotic disease, antithrombotic treatments for cardioembolism, and the use of antiplatelet agents for noncardioembolic stroke. Further recommendations are provided for the prevention of recurrent stroke in a variety of other specific circumstances, including arterial dissections, patent foramen ovale, hyperhomocysteinemia, hypercoagulable states, sickle cell disease, cerebral venous sinus thrombosis, stroke among women, particularly with regard to pregnancy and the use of postmenopausal hormones, the use of anticoagulation after cerebral hemorrhage, and special approaches for the implementation of guidelines and their use in high-risk populations. [
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3. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 3, 2007, No. 2 , , ,

Cerebral venous sinus thrombosis.
Allroggen H, Abbott RJ. Cerebral venous sinus thrombosis.
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acute dural sinus thrombosis.
acute dural sinus thrombosis.
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Magnetic resonance imaging of cerebral venous sinus thrombosis.
Connor SE, Jarosz JM. Magnetic resonance imaging of cerebral venous sinus thrombosis.
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Clinical features and prognostic factors of cerebral venous sinus thrombosis in a prospective series of 59 patients.
de Bruin S, de Haan R, Stam J. Clinical features and prognostic factors of cerebral venous sinus thrombosis in a prospective series of 59 patients.
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Effect of echo-contrast media on the visualization of transverse sinus thrombosis with transcranial 3-D duplex sonography.
Delcker A, Hаussermann P, Weimar C. Effect of echo-contrast media on the visualization of transverse sinus thrombosis with transcranial 3-D duplex sonography.
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Coagulation studies, factor V Leiden and anticardiolipin antibodies in 40 cases of cerebral venous sinus thrombosis.
Deschiens M, Conard J, Horellou M, Ameri A, Preter M, Chedu F. Coagulation studies, factor V Leiden and anticardiolipin antibodies in 40 cases of cerebral venous sinus thrombosis.
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Cerebral venous and sinus thrombosis: an update.
upl KM, Masuhr F. Cerebral venous and sinus thrombosis: an update.
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Cerebral venous sinus thrombosis.
Kimber J. Cerebral venous sinus thrombosis.
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Echocontrastenhanced transcranial color-coded sonography for the diagnosis of transverse sinus thrombosis.
Ries S, Steinke W, Neff KW, Hennerici M. Echocontrastenhanced transcranial color-coded sonography for the diagnosis of transverse sinus thrombosis.
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Еndovascular Thrombectomy and Thrombolysis for Severe Cerebral Sinus Thrombosis: A Prospective Study.
Stam J, Majoie C. B.L.M, van Delden OM, van Lienden KP, Reekers JA. Еndovascular Thrombectomy and Thrombolysis for Severe Cerebral Sinus Thrombosis: A Prospective Study.
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Influence of recanalization on outcome in dural sinus thrombosis: A prospective study.
Stolz E, Trittmacher S, Rahimi A, Gerriets T, Ruttger C, Siekmann R, Kaps M. Influence of recanalization on outcome in dural sinus thrombosis: A prospective study.
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R. Dural sinus thrombosis: value of venous MRA for diagnosis and follow up.
R. Dural sinus thrombosis: value of venous MRA for diagnosis and follow up.
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Anticoagulation in Cerebral Venous Sinus Thrombosis: Are We Treating Ourselves?
Wasay M, Kamal AK. Anticoagulation in Cerebral Venous Sinus Thrombosis: Are We Treating Ourselves?
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4. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 2, 2012, No. 2 , , ,

Effect of echo-contrast media on the visualization of transverse sinus thrombosis with transcranial 3-D duplex sonography.
Delker A, Haussermann P, Weimar C. Effect of echo-contrast media on the visualization of transverse sinus thrombosis with transcranial 3-D duplex sonography.
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Coagulation studies, factor leiden a and anticardiolipin antibodies in 40 cases of cerebral venous sinus thrombosis.
Deschiens M, Conard J, Horellow M, Ameri A, Peter M. Coagulation studies, factor leiden a and anticardiolipin antibodies in 40 cases of cerebral venous sinus thrombosis.
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Echocontrast enhancedtranskranial color-coded sonography for the diagnosis of transverse sinus thrombosis.
Ries S, Steinke W, Neff KW, Hennerici M. Echocontrast enhancedtranskranial color-coded sonography for the diagnosis of transverse sinus thrombosis.
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J. Endovascular thrombectomy and thrombolysis for severe cerebral sinus thrombosis: a prospective study.
J. Endovascular thrombectomy and thrombolysis for severe cerebral sinus thrombosis: a prospective study.
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Influence of recanalization on outcome in dural sinus thrombosis: A prospective study.
Stolz E, trittmacher S, Rahimi A, Geriets t, Ruttger C, Siekmann R, kaps M. Influence of recanalization on outcome in dural sinus thrombosis: A prospective study.
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R. Dural sinus thrombosis: value of venous MRA for diagnosis and follow up.
R. Dural sinus thrombosis: value of venous MRA for diagnosis and follow up.
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anticoagulation in cerebral venous sinus thrombosis: are we treating ourselves?
Wasay m, Kamal A. anticoagulation in cerebral venous sinus thrombosis: are we treating ourselves?
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5. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 9, 2013, No. 1 , , ,

In 1947, Putnam, believing that thrombosis of the cerebral veins was a common finding in MS patients published preliminary results of treatment using dicoumarin in MS patients after experiments using induced sinus thrombosis in primates [50].
He coined the term “CCSVI” (chronic cerebrospinal venous insufficiency) in analogy to perivenous inflammation in chronic venous insufficiency of the legs. While Zamboni’s approach does not challenge the commonly accepted understanding of MS immunopathology [37], it does relegate it to the final stage in the disease cascade. According to the “CCSVI” concept, MS pathology starts with intracranial venous stasis based on a proximal obstruction of the main cervical and/or thoracic veins. This leads to perivenous diapedesis of erythrocytes in the white matter with subsequent release of iron, the actual catalyst of the widely known and accepted immune cascade [37]. The theory of venous outflow changes reaches back to the times of Charcot, who in 1868 provided an early histopathological description of perivenous inflammation in MS [16]. In 1947, Putnam, believing that thrombosis of the cerebral veins was a common finding in MS patients published preliminary results of treatment using dicoumarin in MS patients after experiments using induced sinus thrombosis in primates [50]. However, his findings have not been validated or revisited since this time. In 1986, Schelling posed the hypothesis that venous intracranial or intraspinal reflux plays a significant role in the development of MS [53]. Subsequently, Zamboni and colleagues published several studies which were meant to support the “CCSVI” hypothesis [67,69,70]. They applied catheter angiographies in order to demonstrate various extracranial venous outflow obstructions in the internal jugular veins (IJVs) or azygos veins (AVs) [66], and re-
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In the field of neurology, research begun to focus in the mid-nineties of the past century primarily on impaired venous drainage in primarily venous disorders like cerebral venous and sinus thrombosis [12, 55].
Currently, US is one popular technique for imaging the venous system. In the field of neurology, research begun to focus in the mid-nineties of the past century primarily on impaired venous drainage in primarily venous disorders like cerebral venous and sinus thrombosis [12, 55]. Subsequently, primarily non-venous disease entities were studied. In transient global amnesia (TGA) an increased prevalence of IJV valve insufficiency (IJVVI) was seen which occurs in 20-30% of the normal population, but in up to 70% of TGA patients [1, 52, 54]. A significantly increased prevalence of IJVVI was recently also shown for transient monocular blindness [30], leucoaraiosis [15], primary exertional headache [23], primary intracranial hypertension [40] and chronic obstructive pulmonary disease [19]. More recent research data suggest that the venous system may play a considerable role in arterial stroke. Yu and co-workers found that an impaired ipsilateral venous drainage due to a hypoplastic or aplastic lateral sinus (transversus and sigmoid sinus) was accompanied by pronounced infarction leading to higher morbidity and mortality [65].
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Ultrasound, Stroke, Cerebral venous and sinus thrombosis, Brain Tumours, Migraine
Ultrasound, Stroke, Cerebral venous and sinus thrombosis, Brain Tumours, Migraine
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6. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 9, 2013, No. 2 , , ,

cerebral venous and sinus thrombosis.
The cerebral venous outflow has been neglected for many years in neurology. Considering Neurosonology a first interest started with primarily venous disorders, e.g. cerebral venous and sinus thrombosis. In the last few years venous ultrasound analysis was performed in a variety of other neurological disorders like dural fistulas, transient global amnesia and even in acute arterial stroke. In multiple sclerosis a chronic impaired cerebral venous outflow has claimed to be the starting point of a cascade leading to the disease. This concept has not been reproduced by groups with a longstanding experience in venous duplex sonography. However, the debates increased the scientific interest on the venous side of the cerebral circulation and its outflow.
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7. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 11, 2015, No. 2 , , ,

cerebral venous sinus thrombosis.
cerebral venous sinus thrombosis.
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8. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 12, 2016, No. 1 , , ,

Cavernous sinus thrombosis may be septic or aseptic, which requires further study of a complete blood count (CBC) with differential count (CCA), ESR and C – reactive protein.
thrombosis of the cavernous sinus, whose clinical presentation and course resembles TolosaHunt syndrome. Cavernous sinus thrombosis may be septic or aseptic, which requires further study of a complete blood count (CBC) with differential count (CCA), ESR and C – reactive protein. Those studies help to distinguish from collagenosis. In light of other processes such as retrobulbar orbital pseudotumor, lymphoma, metastasis of unidentified carcinoma requires study of tumor markers (carcinoembryonic antigen and prostate specific antigen). To exclude endocrine ophthalmoplegia is necessary to test
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9. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 12, 2016, No. 2 , , ,

sinus thrombosis, superior ophthalmic vein thrombosis
sinus thrombosis, superior ophthalmic vein thrombosis
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On the other hand infections of dental origin and teeth extractions in the maxilla are well-known triggers for cavernous sinus thrombosis, sometimes with extension to superior ophthalmic veins.
С-reactive protein. On the other hand infections of dental origin and teeth extractions in the maxilla are well-known triggers for cavernous sinus thrombosis, sometimes with extension to superior ophthalmic veins. Since our patient had teeth extractions three days prior to SOVT symptoms onset, this probably was the triggering factor of bilateral SOVT. She had also plasma von Willebrand factor antigen (vWfAg) almost 4-fold above normal values. Plasma concentrations of vWfAg are significantly elevated in patients with acute infectious diseases [12]. This is another proof that the infection was a SOVT trigger in our patient.
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MRl is sensitive even in early stages of the disease, and is recommended when there is a suspicion of SOVT or cavernous sinus thrombosis.
Patients with SOVT may complain of facial and orbital pain and swelling, double and decreasedblurred vision. Сlinical findings depend on the specific etiology and may include proptosis, chemosis, ophthalmoplegia, and ptosis. lf the optic nerve is affected by compression, clinical sigs of optic neuropathy may occur, such as reduced visual acuity, abnormal color vision, and relative afferent papillary defect [1, 2]. ln this case, the patient presented with clinical features consistent with an orbital process. Before additional diagnostics, we suspected Tolosa-Hunt syndrome, but after the MRl we diverted the examination to SOVT. MRl is sensitive even in early stages of the disease, and is recommended when there is a suspicion of SOVT or cavernous sinus thrombosis. MRl may demonstrate a dilated SOV, and extraocular muscle enlargement [2, 9]. ln our case, orbital MR examination was of crucial importance for the diagnosis. Some authors suggest Doppler imaging with ultrasound to confirm the lack of flow in the SOV [6]. We performed an US Doppler examination on the 6th day of treatment introduction which helped us in establishing the definitive diagnosis of SOVT and also monitoring the positive therapy effects. On the same day we performed a control СT of the brain and orbits which was in compliance with the US findings and the good early clinical outcome.
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On the other hand, if not treated, SOVT can progress to cavernous sinus thrombosis.
The appropriate management of SOVT depends on the etiology. 1n all cases empiric treatment with broad spectrum antibiotics is recommended, because infection is one of the most common causes of SOVT; later on antibiotics could be changed according to the antibiogram. Antibiotics should be given for 2 more weeks after clinical resolution, because pathogens can be located and sequestered within the thrombus [14]. Since in our case maxillary teeth extractions were the probable trigger factor for SOVT, we started treatment with dual broad spectrum antibiotics, which fortunately proved to be successful. The role of anticoagulants in SOVT cases is unclear. On the other hand, if not treated, SOVT can progress to cavernous sinus thrombosis. Although the use of anticoagulant therapy is controversial, many authors suggest dose-adjusted intravenous heparin applications if there are no contraindications, such as risk of intracranial or any other hemorrhage [15]. The EFNS guidelines for treatment of cerebral venous and sinus thrombosis in adults recommend body weightadjusted subcutaneous LMWH or dose-adjusted intravenous heparin use with an at least doubled activated partial thromboplastin time [4]. A metaanalysis comparing the efficacy of adjusted-dose unfractionated heparin and fixed-dose subcutaneous LMWH for extracerebral venous thromboembolism found superiority for LMWH and significantly less major bleeding complications [4, 16]. Guided by these recommendations we started the therapy with LMWH. Einhapul et al.
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The EFNS guidelines for treatment of cerebral venous and sinus thrombosis in adults recommend body weightadjusted subcutaneous LMWH or dose-adjusted intravenous heparin use with an at least doubled activated partial thromboplastin time [4].
Antibiotics should be given for 2 more weeks after clinical resolution, because pathogens can be located and sequestered within the thrombus [14]. Since in our case maxillary teeth extractions were the probable trigger factor for SOVT, we started treatment with dual broad spectrum antibiotics, which fortunately proved to be successful. The role of anticoagulants in SOVT cases is unclear. On the other hand, if not treated, SOVT can progress to cavernous sinus thrombosis. Although the use of anticoagulant therapy is controversial, many authors suggest dose-adjusted intravenous heparin applications if there are no contraindications, such as risk of intracranial or any other hemorrhage [15]. The EFNS guidelines for treatment of cerebral venous and sinus thrombosis in adults recommend body weightadjusted subcutaneous LMWH or dose-adjusted intravenous heparin use with an at least doubled activated partial thromboplastin time [4]. A metaanalysis comparing the efficacy of adjusted-dose unfractionated heparin and fixed-dose subcutaneous LMWH for extracerebral venous thromboembolism found superiority for LMWH and significantly less major bleeding complications [4, 16]. Guided by these recommendations we started the therapy with LMWH. Einhapul et al. suggest maintaining an elevated РTT in SOVT patients, but no more than
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EFNS guideline on the treatment of cerebral venous and sinus thrombosis in adult patients.
upl K, Stam J, Bousser MG, De Bruijn SF, Ferro JM, Martinelli I, Masuhr F. European Federation of Neurological Societies. EFNS guideline on the treatment of cerebral venous and sinus thrombosis in adult patients.
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