Search for 'transcranial ultrasound'

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21 results of 9 texts with exact phrase : 'transcranial ultrasound'.

1. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 1, 2005, No. 2 , , ,

Duplex and transcranial ultrasound examination is most often used in everyday practice.
also used: duplex ultrasound (DUS), computed tomography angiography (CTA) and contrast enhanced magnetic resonance angiography (CEMRA). Duplex and transcranial ultrasound examination is most often used in everyday practice. It is less expensive, reliable and suitable for bedside diagnosis. The most common sites for plaque formation – the origin of the common carotid artery (12.4%), the internal carotid artery (ICA) just above the bifurcation (22.3%), the origin of the middle cerebral artery (4.1%) and the basilar artery (8.7%) – can be examined in large majority of patients.
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2. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 2, 2006, No. 2 , , ,

(Combined Lysis of Thrombus in Brain Ischemia using transcranial Ultrasound and Systematic TPA) [5, 10] е изследване, в което са рандомизирани 126 пациенти с ИМИ.
(Combined Lysis of Thrombus in Brain Ischemia using transcranial Ultrasound and Systematic TPA) [5, 10] е изследване, в което са рандомизирани 126 пациенти с ИМИ. То доказа, че продължителното ТДС мониториране (общо 2 часа) чрез 2-МHz диагностична сонда, с мощност 750 mW, постоянни ъгъл и местоположение на инсонация, при дълбочина – мястото с найлош остатъчен ток по TIBI скалата, има положителен ефект върху първичните крайни цели. Не установява повишаване на честотата на хеморагичните мозъчни усложнения при ТЛ на болни с ИМИ и демонстрира тенденция за подобро възстановяване след ИМИ спрямо болните с плацебо [5]. Резултатите от изследването на Alexandrov и сътр. показват, че 2-часовото ТДС мониториране на СМА при болни с ИМИ е без сериозни странични ефекти и усилва индуцираната от t-PA реканализиция (на 2-я час
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Transcranial ultrasound-improved thrombolysis: diagnostic vs.
Behrens S, Spengos K, Daffertshofer M, Schroeck H, Dempfle CE, Hennerici M. Transcranial ultrasound-improved thrombolysis: diagnostic vs. therapeutic ultrasound.
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In Vitro Models for Assessing Transcranial Ultrasound-Enhanced Thrombolysis* Response.
Meairs S, Dempfle C-E, Pfaffenberger S, Speidl WS, Wojta J, Gottsauner-Wolf, M. In Vitro Models for Assessing Transcranial Ultrasound-Enhanced Thrombolysis* Response.
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3. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 6, 2010, No. 1 , , ,

Safety and doseescalation study design of Transcranial Ultrasound in Clinical SONolysis for acute ischemic stroke: the TUCSON Trial.
Barreto AD, Sharma VK, Lao AY, Schellinger PD, Amarenco P, Sierzenski P, Alexandrov AV, Molina CA. Safety and doseescalation study design of Transcranial Ultrasound in Clinical SONolysis for acute ischemic stroke: the TUCSON Trial.
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4. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 6, 2010, No. 2 , , ,

Transcranial Ultrasound Course
Transcranial Ultrasound Course
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5. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 9, 2013, No. 2 , , ,

Transcranial Ultrasound – Experimental, Clinical and Functional
Poster Session I–3. Transcranial Ultrasound – Experimental, Clinical and Functional
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Safety of lowfrequency transcranial ultrasound in permanent middle cerebral artery occlusion in spontaneously hypertensive rats.
Wang Z, Fukuda T, Azuma T, Furuhata H. Safety of lowfrequency transcranial ultrasound in permanent middle cerebral artery occlusion in spontaneously hypertensive rats.
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Transcranial Ultrasound – Experimental, Clinical and Functional
Poster Session I–3. Transcranial Ultrasound – Experimental, Clinical and Functional
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6. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 10, 2014, No. 2 , , ,

Neuropsychological and behavioral examination and evaluation, Neurosonology (Extracranial/Transcranial ultrasound, Temporal arteries sonography, Orbita ultrasonography), Ultrasound examination of the brain parenchyma (basal ganglia), Stroke management (including thrombolytic therapy and sonothombolysis).
Neuropsychological and behavioral examination and evaluation, Neurosonology (Extracranial/Transcranial ultrasound, Temporal arteries sonography, Orbita ultrasonography), Ultrasound examination of the brain parenchyma (basal ganglia), Stroke management (including thrombolytic therapy and sonothombolysis).
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While transcranial ultrasound has very high specificity, sensitivity and negative predictive value, it has only modest positive predictive value, thus requiring confirmation by other imaging modality such as CTA or conventional cerebral angiography.
2) and dampened flow signal (TIBI 3) are criteria for distal occlusion. Indirect criteria of intracranial arterial occlusion comprise high resistance in the feeding vessel or in the proximal segment of the occluded vessel, flow diversion and signs of collateralization. Analogously to intracranial occlusion, intracranial stenosis criteria are direct and indirect. Direct criteria include progressive focal increase of lood flow velocities in 2 50% stenosis or paradoxical velocity decrease with very severe stenosis, near-occlusion or diffuse intracranial disease. Indirect criteria, which are present only in very severe stenosis (>80%), are the same as for occlusion: high resistance in the feeding vessel or in the proximal segment of the stenotic vessel, flow diversion and signs of collateralization. While transcranial ultrasound has very high specificity, sensitivity and negative predictive value, it has only modest positive predictive value, thus requiring confirmation by other imaging modality such as CTA or conventional cerebral angiography.
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Transcranial ultrasound can surely help by studying collaterals, testing for vasomotor reactivity and detecting emboli.
Once the anatomical diagnosis of an intracranial stenosis is made, it is crucial to understand the functional significance and the hemodynamic effects of the stenosis. Transcranial ultrasound can surely help by studying collaterals, testing for vasomotor reactivity and detecting emboli. In fact TCD/TCCS can provide real-time information on collateral flow and in case of vessel obstruction, activation of collateral pathways is very important for the clinical outcome of the patient. A complete circle of Willis and the possibility to activate primary collaterals (anterior communicating artery, posterior communicating artery) or secondary collaterals (ophthalmic artery, leptomeningeal arteries) reduces the risk of hemodynamic ischemic stroke. Sometimes we see a compensatory increase of blood flow velocity in the donor vessel due to recruitment of collaterals by vasodilation in tissues with compromised perfusion. This is called flow diversion and represents a natural steal by vessels distal to an arterial occlusion.
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Transcranial ultrasound is the only diagnostic method that can detect clinically silent emboli; this requires continuous monitoring of the major intracranial arteries and according to the current consensus the duration of the monitoring should be at least one hour.
Transcranial ultrasound is the only diagnostic method that can detect clinically silent emboli; this requires continuous monitoring of the major intracranial arteries and according to the current consensus the duration of the monitoring should be at least one hour. Microembolic signal (MES) detection identifies patients who are at higher risk of atheroembolic stroke, thus allowing to select those patients who could benefit from a more aggressive treatment. MES are also valid surrogate markers for verifying antithrombotic efficacy and a key for individualized stroke medicine.
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In these patients, angioplasty alone or in combination with new stent types might still be an option, and transcranial ultrasound can quickly assess vessel patency by recording intracranial arterial hemodynamics changes post-operatively.
cium channel blockers for vasospasm, antiplatelet agents for dissection, immunosuppressants for vasculitis, intensive risk factor management and dual antiplatelet treatment for ICAS. In patients with recurrent symptoms despite best medical therapy, ultrasound can detect a possible progression of the stenosis, exclude a branch occlusion, check for a possible increase of the embolic count downstream. The SAMMPRIS study has shown that early aggressive medical therapy is better than stenting for prevention of recurrent stroke. Nevertheless, there are subgroups of patients who remain at high risk of stroke despite aggressive medical therapy. In these patients, angioplasty alone or in combination with new stent types might still be an option, and transcranial ultrasound can quickly assess vessel patency by recording intracranial arterial hemodynamics changes post-operatively. In particular, due to the metal composition of the stent, TCCS can clearly display the stent, thereby determining the location and shape of it. When the treatment is effective, there is a significant and immediate decrease of blood flow velocities; after about a week, owing to the reshaping of the stent and vascular remodeling, there is a further improvement in hemodynamics with velocity values declining toward normal. A regular follow-up of these patients is advisable in order to confirm the efficacy of stenting and to detect residual stenosis or in-stent restenosis. Finally, it is important to underline that
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Overall transcranial ultrasound provides accurate information on cerebral hemodynamics and represents the ideal modality for following disease progression and therapeutic effects.
blood flow velocities in a stented vessel are higher compared to a non-stented vessel; consequently, when verifying treatment efficacy this has to be taken into account to avoid overestimation of residual stenosis or misdiagnosis of in-stent restenosis. Overall transcranial ultrasound provides accurate information on cerebral hemodynamics and represents the ideal modality for following disease progression and therapeutic effects.
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Transcranial ultrasound in clinical sonothrombolysis (TUCSON) trial.
Molina CA, Barreto AD, Tsivgoulis G, et al. Transcranial ultrasound in clinical sonothrombolysis (TUCSON) trial.
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L. Intracranial clot lysis with intravenous microbubbles and transcranial ultrasound in swine.
L. Intracranial clot lysis with intravenous microbubbles and transcranial ultrasound in swine.
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7. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 12, 2016, No. 2 , , ,

Non-invasive transcranial ultrasound therapy based on a 3D CT scan: protocol validation and in vitro results.
Marquet F, Pernot M, Aubry J-F, Montaldo G, Marsac L, Tanter M, Fink M. Non-invasive transcranial ultrasound therapy based on a 3D CT scan: protocol validation and in vitro results.
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8. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 13, 2017, No. 2 , , ,

Doctoral thesis in medicine: University of Lausanne, Switzerland; Swiss federal Diploma of Medicine, University of Lausanne, Switzerland; Swiss specialty board certificate in cerebrovascular diseases, Swiss Society of Clinical Neurophysiology FMH (2004); Swiss specialty board certificate in Neurology Swiss Society of Neurology FMH (2005); Certificate of management for senior doctors of the University Hospital of Geneva HUG (2007); International Certificate in Neurosonology ESNCH (2008); Privat Docent Thesis “Transcranial ultrasound in stroke and its new developments” Medical Faculty University of Geneva (2009); Professor of Neurology of the Faculty of Medicine in Geneva, Switzerland (2016).
Doctoral thesis in medicine: University of Lausanne, Switzerland; Swiss federal Diploma of Medicine, University of Lausanne, Switzerland; Swiss specialty board certificate in cerebrovascular diseases, Swiss Society of Clinical Neurophysiology FMH (2004); Swiss specialty board certificate in Neurology Swiss Society of Neurology FMH (2005); Certificate of management for senior doctors of the University Hospital of Geneva HUG (2007); International Certificate in Neurosonology ESNCH (2008); Privat Docent Thesis “Transcranial ultrasound in stroke and its new developments” Medical Faculty University of Geneva (2009); Professor of Neurology of the Faculty of Medicine in Geneva, Switzerland (2016).
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deep brain stimulation, substantia nigra, transcranial ultrasound
deep brain stimulation, substantia nigra, transcranial ultrasound
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9. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 14, 2018, No. 2 , , ,

By means of transcranial ultrasound sonography (TCD, 2 MHz probe, head fixation) of the middle cerebral artery (MCA), the cerebral blood flow velocity (CBFV) was recorded and the mean CBFV was calculated.
We investigated 25 consecutive patients a few month after implantation of a LVAD in our rehabilitation department. By means of transcranial ultrasound sonography (TCD, 2 MHz probe, head fixation) of the middle cerebral artery (MCA), the cerebral blood flow velocity (CBFV) was recorded and the mean CBFV was calculated. Patients were first recorded in supine positon and then brought into standing position. We measured the mean CBFV after standing up and registered any symptoms ranging from dizziness to even postural control failures.
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