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42 results of 15 texts with exact phrase : 'transient ischemic attack'.

1. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, Vol. 1, 2005 , , ,

Risk of stroke, transient ischemic attack, and vessel occlusion before endarterectomy in patients with symptomatic severe carotid stenosis.
3, Blaser T, Hofmann K, Buerger T, Effenberg O, Wallesch C, Goertler M. Risk of stroke, transient ischemic attack, and vessel occlusion before endarterectomy in patients with symptomatic severe carotid stenosis.
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2. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 1, 2005, No. 2 , , ,

An important cause of transient ischemic attack (TIA) and stroke is atherosclerotic carotid artery stenosis.
An important cause of transient ischemic attack (TIA) and stroke is atherosclerotic carotid artery stenosis. It accounts for about 20% of cases of brain infarction and has the highest recurrent stoke risk compared to all other subtypes of stroke [2, 3]. Therefore, rapid intervention in this patient group is needed and they should be managed efficiently to minimize the incidence of stroke.
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M. Ultrasonic correlates of carotid atherosclerosis in transient ischemic attack and stroke.
M. Ultrasonic correlates of carotid atherosclerosis in transient ischemic attack and stroke.
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Microembolic signals and risk of early recurrence in patients with stroke or transient ischemic attack.
raud G. Microembolic signals and risk of early recurrence in patients with stroke or transient ischemic attack.
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3. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 3, 2007, No. 1 , , ,

A reappraisal of the definition and pathophysiology of the transient ischemic attack.
Hadjiev D, Mineva P. A reappraisal of the definition and pathophysiology of the transient ischemic attack.
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Krol AL, Coutts SB, Simon JE et al, VISION Study Group: Perfusion MRI abnormalities in speech or motor transient ischemic attack patients.
Krol AL, Coutts SB, Simon JE et al, VISION Study Group: Perfusion MRI abnormalities in speech or motor transient ischemic attack patients.
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Restrepo L, Jacobs MA, Barker PB, Wityk R: Assessment of transient ischemic attack with diffusionand perfusionweighted imaging.
Restrepo L, Jacobs MA, Barker PB, Wityk R: Assessment of transient ischemic attack with diffusionand perfusionweighted imaging.
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Association of transient ischemic attack/stroke symptoms assessed by standardized questionnaire and algorithm with cerebrovascular risk factors and carotid artery wall thickness.
Chambless LE, Shahar E, Sharrett AR, Heiss G, Wijnberg L, Paton CC, Sorlie P, Toole JF. Association of transient ischemic attack/stroke symptoms assessed by standardized questionnaire and algorithm with cerebrovascular risk factors and carotid artery wall thickness. The ARIC Study, 1987-1989.
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4. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 3, 2007, No. 2 , , ,

or Transient Ischemic Attack
or Transient Ischemic Attack
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Prevalence of significant carotid artery stenosis in patients with transient ischemic attack.
Rappeport Y, Simonsen L, Christianen H, Boysen G. Prevalence of significant carotid artery stenosis in patients with transient ischemic attack.
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Guidelines for prevention of stroke in patients with ishemic stroke or transient ischemic attack.
Sacco R, Adams R, Albert G, Albert M, Benavente O, Furie K, Goldstein LB, Gorelick P, Halperin J, Harbaugh R, Johnston SC, Katzan I, Kelly-Hayes M, Kenton EJ, Marks M, Schwamm LH, Tomsick T. Guidelines for prevention of stroke in patients with ishemic stroke or transient ischemic attack. A statement for Healthcare professionals from the American Heart Association/American Stroke Association Council on Stroke. C0-sponsored by the Council on Cardiovascular Radiology and Intervention.
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A comprehensive review of the published guidelines and recommendations on the prevention of ischemic stroke among survivors of ischemic stroke or transient ischemic attack is presented.
A comprehensive review of the published guidelines and recommendations on the prevention of ischemic stroke among survivors of ischemic stroke or transient ischemic attack is presented. Evidence-based recommendations are included for the control of risk factors, interventional approaches for atherosclerotic disease, antithrombotic treatments for cardioembolism, and the use of antiplatelet agents for noncardioembolic stroke. Further recommendations are provided for the prevention of recurrent stroke in a variety of other specific circumstances, including arterial dissections, patent foramen ovale, hyperhomocysteinemia, hypercoagulable states, sickle cell disease, cerebral venous sinus thrombosis, stroke among women, particularly with regard to pregnancy and the use of postmenopausal hormones, the use of anticoagulation after cerebral hemorrhage, and special approaches for the implementation of guidelines and their use in high-risk populations. [
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Guidelines for Prevention of Stroke in Patients With Ischemic Stroke or Transient Ischemic Attack: A Statement for Healthcare Professionals From the American Heart Association/American Stroke Association Council on Stroke: Co-Sponsored by the Council on Cardiovascular Radiology and Intervention: The American Academy of Neurology affirms the value of this guideline.
* RL Sacco, R Adams, G Albers, MJ Alberts, O Benavente, K Furie, LB Goldstein, P Gorelick, J Halperin, R Harbaugh, SC Johnston, I Katzan, M Kelly-Hayes, EJ Kenton, M Marks, LH Schwamm, T Tomsick. Guidelines for Prevention of Stroke in Patients With Ischemic Stroke or Transient Ischemic Attack: A Statement for Healthcare Professionals From the American Heart Association/American Stroke Association Council on Stroke: Co-Sponsored by the Council on Cardiovascular Radiology and Intervention: The American Academy of Neurology affirms the value of this guideline. Stroke 37, 2006:577-617.
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5. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 4, 2008, No. 1 , , ,

Dissection of Aorta and Both Carotid Arteries in a Patient with Transient Ischemic Attack
Dissection of Aorta and Both Carotid Arteries in a Patient with Transient Ischemic Attack
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We describe а 62-year old man with transient ischemic attack in the right middle cerebral artery and dissection of aorta ascendens and descendens.
We describe а 62-year old man with transient ischemic attack in the right middle cerebral artery and dissection of aorta ascendens and descendens. The precise diagnosis was made by Dopper, duplex sonography, echocardiography and spiral multislice computer tomography. Hemodynamic changes in the basal cerebral arteries in aorta dissection and indications for thrombolysis in patients with cerebral infarctions due to arterial dissection are discussed.
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6. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 6, 2010, No. 1 , , ,

Guidelines for Management of Ischemic Stroke and Transient Ischemic Attack. 2008.
European Stroke Organization (ESO) Executive Committee and the ESO Writing Committee. Guidelines for Management of Ischemic Stroke and Transient Ischemic Attack. 2008.
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The early periprocedural complications were: one ipsilateral stroke (1.9%), one transient ischemic attack, one myocardial infarction (1.9%), one death (1.9%) and one complication at the puncture site.
A succesfull stenting was achieved in 50 cases (98%). The early periprocedural complications were: one ipsilateral stroke (1.9%), one transient ischemic attack, one myocardial infarction (1.9%), one death (1.9%) and one complication at the puncture site. The combined index of stroke/death/MI was found in 2 cases (3.9%). The late 18-month-follow-up showed 1 stroke, 1 death (not related to the procedure) and 1 in-stent restenosis.
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ABCD score, ABCD(2) score, long term risk, short term risk, stroke, transient ischemic attack
ABCD score, ABCD(2) score, long term risk, short term risk, stroke, transient ischemic attack
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Transient ischemic attack (TIA) is a medical emergency and must be treated as an „acute cerebrovascular accident”.
Transient ischemic attack (TIA) is a medical emergency and must be treated as an „acute cerebrovascular accident”. The risk of subsequent stroke both soon and long after TIA is significantly higher compared with other the population group. Existing clinical scales, as California score, ABCD score and unified ABCD(2) score are easy of access markedly available to identify practical risk management and allow selection of emergency patients with TIA. Independent interpretation of accompanying risk factors and integrated data as a result of specialized biochemical tests, ultrasound diagnostics and neuroimaging methods provide more detailed picture of the prognosis and probability. The modern concept of TIA as a condition requiring emergency has found practical realization in the creation of so-called “TIA Clinic”.
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Higher ABCD2 score predicts patients most likely to have true transient ischemic attack.
Higher ABCD2 score predicts patients most likely to have true transient ischemic attack.
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T. The duration of symptoms in transient ischemic attack.
T. The duration of symptoms in transient ischemic attack.
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Stroke Risk After Transient Ischemic Attack in a Population-Based Setting.
Lisabeth LD, Ireland KJ, Risser JMH, Brown DL, Smith AM, Garcia NM, Morgenstern LB. Stroke Risk After Transient Ischemic Attack in a Population-Based Setting.
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Early Stroke Risk After Transient Ischemic Attack Among Individuals With Symptomatic Intracranial Artery Stenosis.
Ovbiagele B, Cruz-Flores S, Lynn MJ, Chimowitz MI, for the Warfarin-Aspirin Symptomatic Intracranial Disease (WASID) Study Group. Early Stroke Risk After Transient Ischemic Attack Among Individuals With Symptomatic Intracranial Artery Stenosis.
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Patterns and predictors of early risk of recurrence after transient ischemic attack with respect to etiologic subtypes.
Purroy F, Montaner J, Molina CA, Delgado P, Ribo M, AlvarezSabin J. Patterns and predictors of early risk of recurrence after transient ischemic attack with respect to etiologic subtypes.
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Population-Based Study of ABCD2 Score, Carotid Stenosis, and Atrial Fibrillation for Early Stroke Prediction After Transient Ischemic Attack.
Sheehan OC, Kyne L, Kelly LA, Hannon N, Marnane M, Merwick A, McCormack PM, Duggan J, Moore A, Moroney J, Daly L, Harris D, Horgan G, Williams EB, Kelly PJ. Population-Based Study of ABCD2 Score, Carotid Stenosis, and Atrial Fibrillation for Early Stroke Prediction After Transient Ischemic Attack. The North Dublin TIA Study.
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Transient ischemic attack, a medical emergency.
Uchiyama S. Transient ischemic attack, a medical emergency.
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7. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 6, 2010, No. 2 , , ,

Guidelines for prevention of stroke in patients with ishemic stroke or transient ischemic attack.
Sacco R, Adams R, Albert G, Albert M, Benavente O, Furie K, Goldstein LB, Gorelick P, Halperin J, Harbaugh R, Johnston SC, Katzan I, Kelly-Hayes M, Kenton EJ, Marks M, Schwamm LH, Tomsick T. Guidelines for prevention of stroke in patients with ishemic stroke or transient ischemic attack. A statement for Healthcare professionals from the American Heart Association/American Stroke Association Council on Stroke. C0-sponsored by the Council on Cardiovascular Radiology and Intervention.
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8. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 7, 2011, No. 1 , , ,

patients with transient ischemic attack and stroke: a prospective study of 59 patients.
patients with transient ischemic attack and stroke: a prospective study of 59 patients.
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„Correlation between ABCD(2) score, cerebral vascular territory and dyslipidemia in patients with transient ischemic attack“
„Correlation between ABCD(2) score, cerebral vascular territory and dyslipidemia in patients with transient ischemic attack“
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9. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 7, 2011, No. 2 , , ,

Guidelines for the Prevention of Stroke in Patients With Stroke or Transient Ischemic Attack: A Guideline for Healthcare Professionals From the American Heart Association/American Stroke Association.
Furie KL, Kasner SE, Adams RJ, Albers GW, Bush RL, Fagan SC, Halperin JL, Johnston SC, Katzan I., Kernan WN et al. Guidelines for the Prevention of Stroke in Patients With Stroke or Transient Ischemic Attack: A Guideline for Healthcare Professionals From the American Heart Association/American Stroke Association.
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10. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 9, 2013, No. 2 , , ,

Data from all patients who underwent interventional procedures during the period 2003–2012, caused by a symptomatic (transient ischemic attack or stroke) stenosis of a major intracranial artery, were extracted from a stent data base.
Data from all patients who underwent interventional procedures during the period 2003–2012, caused by a symptomatic (transient ischemic attack or stroke) stenosis of a major intracranial artery, were extracted from a stent data base. Technical success, clinical outcome, the rate of instent restenosis (ISR) and recurrent stroke as well as prognosis were defined as primary objectives. Examinations were at 24 hours and 6 months after the procedure, as well as the last consultation of the stroke outpatient clinic.
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Ipsilateral stroke or transient ischemic attack occurred in 3.2% during the first 24 hours, in in 6.5% within 30 days, and in 15.1% until 12 months after intervention.
89 patients (20 female, 69 male; mean age 67.3 years) with 93 symptomatic intracranial stenoses were treated by interventional procedures. Technical success rate was 98,9%. After a mean follow up time of 2.6 years, 78.8% showed a good clinical outcome according to the modified Rankin Scale (mRS) scores of 0–2, while 15.7% were disabled (mRS 3–5). 5.6% deceased, none of them within the first 30 days after the initial intervention. A 24-hours post procedure ISR – rate of 7.5% (7 patients), a 6-months rate of 16.5% (14 patients) and a long-term rate of 13.0% (12 patients) was diagnosed. Ipsilateral stroke or transient ischemic attack occurred in 3.2% during the first 24 hours, in in 6.5% within 30 days, and in 15.1% until 12 months after intervention.
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The most common types of CVDs are ischemic stroke, transient ischemic attack, hemorrhagic stroke and vascular dementia.
Cerebrovascular diseases (CVD) represent conditions which occur as a result of changes in blood vessels of the brain, as well as the vessels supplying the brain. The most common types of CVDs are ischemic stroke, transient ischemic attack, hemorrhagic stroke and vascular dementia. CVDs affect millions of people worldwide, regardless of age, and represent a group of very important medical and social problems. Therefore, their prevention is becoming an imperative. Risk factors, such as age, gender, genetic factors, hypertension, diabetes mellitus, hypercholsterolemia, atrial fibrillation, orlifestyle,are causing changes of vessel walls which lead to CVD. Early changes of the blood vessel wall can be detected by early ultrasound screening methods which allow us to detect changes before the disease becomes clinically evident. Intracranial hemodynamics can be assessed by Transcranial Doppler Sonography (TCD), functional TCD with various functional tests, and TCD detection of cerebral emboli.
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The risk of transient ischemic attack and stroke following transfemoral aortic valve implantation (TAVI) is 2-10% and is even higher considering silent ischemic brain injury.
The risk of transient ischemic attack and stroke following transfemoral aortic valve implantation (TAVI) is 2-10% and is even higher considering silent ischemic brain injury. Periprocedural microembolic signals (MES) and hemodynamic changes (HC) may be considered physiopathological markers of such events. We show neurosonological and neuroradiological data about a patient submitted to TAVI.
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11. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 10, 2014, No. 2 , , ,

Fabry disease patients, without prior history of stroke or transient ischemic attack, had disturbed neurovascular coupling in the visual cortex.
Functional TCD can also be useful for evaluating presymptomatic and even prelesional subjects with genetic diseases afecting the endothelial function. Fabry disease patients, without prior history of stroke or transient ischemic attack, had disturbed neurovascular coupling in the visual cortex.
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The authors concluded that Fabry disease patients of both genders, without prior history of stroke or transient ischemic attack, may have disturbed neurovascular coupling in the visual cortex, as well as decreased resting posterior cerebral artery BFV.
Functional transcranial Doppler can also be useful for evaluating presymptomatic and even prelesional subjects with genetic diseases affecting the endothelial function. As cerebrovascular disease is known to progress asymptomatically in the early stages of Fabry disease, a cohort of patients from families with the classical phenotype were studied with functional transcranial Doppler. The authors concluded that Fabry disease patients of both genders, without prior history of stroke or transient ischemic attack, may have disturbed neurovascular coupling in the visual cortex, as well as decreased resting posterior cerebral artery BFV. These findings support the role of functional TCD, along with duplex ultrasound and MR techniques, in the evaluation of these patients, since early stages of disease [4]. Finally, interesting findings seem to show that cerebral blood flow regulation may reflect the neurological dysfunction caused by cerebral microvascular disease [22, 23]. Data from the MOBILIZE Boston study shows that changes in CBF velocity responses to an N-Back task to study the NVC was significantly associated with gait speed and that subjects with higher NVC were able to suppress the negative relationship between white matter hyperintensities and gait speed [23]. A better understanding of the relationship between cerebral hemodynamics and structural changes in the aging brain is an essential step towards identifying preventive and therapeutic strategies
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12. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 11, 2015, No. 2 , , ,

Atrial septal defect and consequently the existence of right-to-left cardiac shunt (RLS) is a very common etiological factor in the onset of ischemic stroke and transient ischemic attack (TIA) in younger patients.
Atrial septal defect and consequently the existence of right-to-left cardiac shunt (RLS) is a very common etiological factor in the onset of ischemic stroke and transient ischemic attack (TIA) in younger patients. Since the contrast transesophageal echocardiography (c-TEE) is considered the “gold” standard for right-to-left cardiac shunt detection, we compared its sensitivity with the sensitivity of contrast-enhanced transcranial Doppler ultrasound (TCD). We also studied the influence of vascular risk factors on TCD parameters.
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Use of Carotid Intima-Media Thickness to Identify Patients With Ischemic Stroke and Transient Ischemic Attack With Low Yield of Cardiovascular Sources of Embolus on Transesophageal Echocardiography.
Ward PR, Lammertin G, Virnich DE et al. Use of Carotid Intima-Media Thickness to Identify Patients With Ischemic Stroke and Transient Ischemic Attack With Low Yield of Cardiovascular Sources of Embolus on Transesophageal Echocardiography.
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13. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 13, 2017, No. 1 , , ,

The European Stroke Organization (ESO) guidelines for management of ischemic stroke and transient ischemic attack recommend the following [36]:
The European Stroke Organization (ESO) guidelines for management of ischemic stroke and transient ischemic attack recommend the following [36]:
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14. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 14, 2018, No. 1 , , ,

– neurological damage leading to brain death, non-fatal stroke, transient ischemic attack.
– neurological damage leading to brain death, non-fatal stroke, transient ischemic attack.
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Some authors determine additional risks such as: females, renal impairment, smoking, ejection fraction under 40%, history of previous ischemic stroke or transient ischemic attack, aortic calcification, prolonged surgery, emergency surgery, surgery for aortocoronary bypass and others [7].
The major risk factors are determined to be carotid stenosis, the presence of insulin-dependent diabetes, and peripheral arterial disease. According to some authors, age of 70 or more is a minor risk factor [7], while others deem old age to be a dominant risk factor [12]. Some authors determine additional risks such as: females, renal impairment, smoking, ejection fraction under 40%, history of previous ischemic stroke or transient ischemic attack, aortic calcification, prolonged surgery, emergency surgery, surgery for aortocoronary bypass and others [7].
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15. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 14, 2018, No. 2 , , ,

The intraluminal carotid thrombosis (ICT) is an infrequent finding in cases of transient ischemic attack or ischemic stroke.
The intraluminal carotid thrombosis (ICT) is an infrequent finding in cases of transient ischemic attack or ischemic stroke. Optimal treatment for patients with symptomatic ICT remains poorly defined. Treatment options include anticoagulation/antiplatelet therapy and/or revascularization procedures.
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