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12 results of 3 texts with exact phrase : 'visual sensory pathways'.

1. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 1, 2005, No. 2 , , ,

Other are a result of definite choroidal, retinal, optic nerve, visual sensory pathways damage, to wit: central retinal artery or branch central retinal artery occlusion, ischemic optic neuropathy, unilateral venous-stasis retinopathy, ischemic ocular syndrome, contralateral homonymous hemianopias and quadrantanopias.
Various ophthalmologic disorders as an isolated independent manifestation or in combination with concomitant neurologic symptomatology can be observed in patients with carotid occlusive disease (carotid stenosis or thrombosis). Some of these symptoms are transient like amaurosis fugax (transient monocular blindness), which is very frequent predictor of the carotid occlusive disease. Other are a result of definite choroidal, retinal, optic nerve, visual sensory pathways damage, to wit: central retinal artery or branch central retinal artery occlusion, ischemic optic neuropathy, unilateral venous-stasis retinopathy, ischemic ocular syndrome, contralateral homonymous hemianopias and quadrantanopias. Their exact interpretation, especially of the transient ophthalmologic symptoms, supports the early diagnosis of carotid pathology and prevents the late definite neurologic and ophthalmologic complications.
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2. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 2, 2006, No. 1 , , ,

of the Visual Sensory Pathways
of the Visual Sensory Pathways
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Neuro-ophthalmological Symptomatology in Patients with Vascular Lesions of the Visual Sensory Pathways
Neuro-ophthalmological Symptomatology in Patients with Vascular Lesions of the Visual Sensory Pathways
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visual sensory pathways
visual sensory pathways
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Vascular lesions of the visual sensory pathways could be an isolated clinical manifestation or be joined to other neurological symptomatology.
Vascular lesions of the visual sensory pathways could be an isolated clinical manifestation or be joined to other neurological symptomatology. The type of the visual field defect, the visual acuity and the ophthalmoscopic appearance of the eye fundi and their dynamics over time are marked with specificity depending on the location of the lesion – prechiasmal, chiasmal or retrochiasmal segment of the visual sensory pathways. The prechiasmal lesions cause monocular field defects (altitudinal, arcuate, central scotoma and others), decreased visual acuity and typical ophthalmoscopic changes. Vascular lesions of the optic chiasm are extremely rare and are characterized by the typical for the chiasmal lesions bitemporal perimetric field defects, decreased visual acuity and descendent optic atrophy. Homonymous hemianopias and quadrantopias, no pupil pathology, normal visual acuity and normal appearance of the eye fundi are typical features of the retrochiasmal lesions of the visual sensory pathways with vascular etiology.
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The type of the visual field defect, the visual acuity and the ophthalmoscopic appearance of the eye fundi and their dynamics over time are marked with specificity depending on the location of the lesion – prechiasmal, chiasmal or retrochiasmal segment of the visual sensory pathways.
Vascular lesions of the visual sensory pathways could be an isolated clinical manifestation or be joined to other neurological symptomatology. The type of the visual field defect, the visual acuity and the ophthalmoscopic appearance of the eye fundi and their dynamics over time are marked with specificity depending on the location of the lesion – prechiasmal, chiasmal or retrochiasmal segment of the visual sensory pathways. The prechiasmal lesions cause monocular field defects (altitudinal, arcuate, central scotoma and others), decreased visual acuity and typical ophthalmoscopic changes. Vascular lesions of the optic chiasm are extremely rare and are characterized by the typical for the chiasmal lesions bitemporal perimetric field defects, decreased visual acuity and descendent optic atrophy. Homonymous hemianopias and quadrantopias, no pupil pathology, normal visual acuity and normal appearance of the eye fundi are typical features of the retrochiasmal lesions of the visual sensory pathways with vascular etiology.
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Homonymous hemianopias and quadrantopias, no pupil pathology, normal visual acuity and normal appearance of the eye fundi are typical features of the retrochiasmal lesions of the visual sensory pathways with vascular etiology.
Vascular lesions of the visual sensory pathways could be an isolated clinical manifestation or be joined to other neurological symptomatology. The type of the visual field defect, the visual acuity and the ophthalmoscopic appearance of the eye fundi and their dynamics over time are marked with specificity depending on the location of the lesion – prechiasmal, chiasmal or retrochiasmal segment of the visual sensory pathways. The prechiasmal lesions cause monocular field defects (altitudinal, arcuate, central scotoma and others), decreased visual acuity and typical ophthalmoscopic changes. Vascular lesions of the optic chiasm are extremely rare and are characterized by the typical for the chiasmal lesions bitemporal perimetric field defects, decreased visual acuity and descendent optic atrophy. Homonymous hemianopias and quadrantopias, no pupil pathology, normal visual acuity and normal appearance of the eye fundi are typical features of the retrochiasmal lesions of the visual sensory pathways with vascular etiology.
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Neuro-ophthalmological symptomatology in vascular visual sensory pathways lesions
Neuro-ophthalmological symptomatology in vascular visual sensory pathways lesions
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Neuro-ophthalmological symptomatology in vascular visual sensory pathways lesions
Neuro-ophthalmological symptomatology in vascular visual sensory pathways lesions
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Neuro-ophthalmological symptomatology in vascular visual sensory pathways lesions
Neuro-ophthalmological symptomatology in vascular visual sensory pathways lesions
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3. NEUROSONOLOGY AND CEREBRAL HEMODYNAMICS, vol. 5, 2009, No. 1 , , ,

vascular lesion with occipital, temporal or parietal localization; In 3 patients – a result of hemorrhagic accident and in the other 2 cases the causes for the visual complaints are AVMs compressing the visual sensory pathways.
vascular lesion with occipital, temporal or parietal localization; In 3 patients – a result of hemorrhagic accident and in the other 2 cases the causes for the visual complaints are AVMs compressing the visual sensory pathways. Thirteen of the examined patients have homonymous hemianopic (complete/incomplete) field defects, 6 of the patients have homonymous quadrantopic or sectoranopic defects and only 1 has a field defect in the pattern of a homonymous scotomata. Ten patients with predominantly occipital localization of the vascular lesion have homonymous hemianopic field defects with “macular sparing” and 3 of the patients have the typical for pregeniculate lesions of the optic tract “macular splitting”. Twelve patients have homonymous field defects with high degree of congruity while the rest of the patients have field defects with lower degree or no congruity at all depending on the localization of the vascular lesion. The CP finding is the only clinical symptom of the cerebrovascular accident in 13 patients. Ten patients are dynamically observed for the period of 1 to 6 months after the onset of the disease.
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for testing different parts of the visual field) helps the precise and early diagnosis and the objective observation of the field defects caused by vascular lesions of the visual sensory pathways.
for testing different parts of the visual field) helps the precise and early diagnosis and the objective observation of the field defects caused by vascular lesions of the visual sensory pathways.
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